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Seong-Gyu Ko

Researcher at Kyung Hee University

Publications -  294
Citations -  4615

Seong-Gyu Ko is an academic researcher from Kyung Hee University. The author has contributed to research in topics: Apoptosis & Cancer. The author has an hindex of 33, co-authored 254 publications receiving 3502 citations.

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Anti-inflammatory effects of Scutellaria baicalensis water extract on LPS-activated RAW 264.7 macrophages.

Seok-Bin Yoon, +9 more
- 07 Sep 2009 - 
TL;DR: Results suggest that SR has anti-inflammatory activity related with its inhibition of NO, cytokine, chemokine, and growth factor production in macrophages.
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Kaempferol induces autophagic cell death via IRE1-JNK-CHOP pathway and inhibition of G9a in gastric cancer cells

Tae Woo Kim, +4 more
- 29 Aug 2018 - 
TL;DR: The findings indicate that kaempferol activates the IRE1-JNK-CHOP signaling from cytosol to nucleus, and G9a inhibition activates autophagic cell death in GC cells, which indicates that kaEMPferol mediates epigenetic change via the inhibition of G9A (HDAC/G9a axis) and also activates autophile cell death.
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World Congress Integrative Medicine & Health 2017: Part one

Carolyn Ee, +776 more
- 30 Jun 2017 - 
TL;DR: This work is presented at the I1 World Congress for Integrative Medicine & Health 2017 a global forum for exploring the future of comprehensive patient care in Washington, DC.
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Use of scopoletin to inhibit the production of inflammatory cytokines through inhibition of the IκB/NF-κB signal cascade in the human mast cell line HMC-1

Phil-Dong Moon, +10 more
- 26 Jan 2007 - 
TL;DR: Results indicate that scopoletin has a potential regulatory effect on inflammatory reactions that are mediated by mast cells.
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Induction of apoptotic cell death by ursolic acid through mitochondrial death pathway and extrinsic death receptor pathway in MDA-MB-231 cells.

Kyung Hun Kim, +5 more
- 11 Sep 2011 - 
TL;DR: The data indicate that ursolic acid could be used as a potential anticancer drug for breast cancer and induce apoptosis through both mitochondrial death pathway and extrinsic death receptor dependent pathway in MDA-MB-231 cells.