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Trevor W. Robbins

Researcher at University of Cambridge

Publications -  1184
Citations -  177352

Trevor W. Robbins is an academic researcher from University of Cambridge. The author has contributed to research in topics: Prefrontal cortex & Cognition. The author has an hindex of 231, co-authored 1137 publications receiving 164437 citations. Previous affiliations of Trevor W. Robbins include Centre national de la recherche scientifique & Massachusetts Institute of Technology.

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Associative processes in addiction and reward. The role of amygdala-ventral striatal subsystems

TL;DR: Understanding of the special role of the ventral striatum is enriched in coordinating the contribution of different functional subsystems to confer flexibility, as well as coherence and vigor, to goal‐directed behavior, through different forms of associative learning.
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Evidence for executive dysfunction in autism.

TL;DR: Evidence for executive dysfunction in autism is discussed in the context of Norman and Shallice's (Centre for Human Information Processing Technical Report 99, 1980) "Supervisory Attentional System" model of frontal function.
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Different types of fear-conditioned behaviour mediated by separate nuclei within amygdala

TL;DR: This work shows that distinct neural systems involving separate amygdaloid nuclei mediate different types of conditioned fear behaviour, and suggests that theories of amygdala function should take into account the roles of discrete amygdala subsystems in controlling different components of integrated emotional responses.
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The cognitive ability of an incident cohort of Parkinson's patients in the UK. The CamPaIGN study

TL;DR: The pattern of cognitive deficits seen among patients using the Mini-Mental State Examination, a pattern recognition task, and the Tower of London task suggests that sub-groups of patients based on cognitive ability might be identifiable even in the early stages of disease, which may reflect regional differences in the underlying neuropathological processes.
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Contrasting mechanisms of impaired attentional set-shifting in patients with frontal lobe damage or Parkinson's disease

TL;DR: The results suggest that the gross set-shifting deficits reported in both frontal lobe patients and patients with Parkinson's disease may involve fundamentally different, though related, cognitive processes, and that these may be differentially affected by medication.