T
Trevor W. Robbins
Researcher at University of Cambridge
Publications - 1184
Citations - 177352
Trevor W. Robbins is an academic researcher from University of Cambridge. The author has contributed to research in topics: Prefrontal cortex & Cognition. The author has an hindex of 231, co-authored 1137 publications receiving 164437 citations. Previous affiliations of Trevor W. Robbins include Centre national de la recherche scientifique & Massachusetts Institute of Technology.
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Neuropsychological and clinical heterogeneity of cognitive impairment and dementia in patients with Parkinson's disease
TL;DR: Remediation and management prospects for cognitive deficits in patients with Parkinson's disease are based on neuropharmacological and cognitive rehabilitation approaches, supplemented by advances in neuroimaging and genetic research.
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Behavioral models of impulsivity in relation to ADHD: Translation between clinical and preclinical studies
TL;DR: This review describes and evaluates some of the current behavioral models of impulsivity developed for use with rodents based on human neuropsychological tests, focusing on the five-choice serial reaction time task, the stop-signal reaction timetask and delay-discounting paradigms.
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The neuropsychology of obsessive compulsive disorder: the importance of failures in cognitive and behavioural inhibition as candidate endophenotypic markers
Samuel R. Chamberlain,Andrew D. Blackwell,Naomi A. Fineberg,Trevor W. Robbins,Barbara J. Sahakian +4 more
TL;DR: It is proposed that neurocognitive indices of inhibitory functions may represent a useful heuristic in the search for endophenotypes in OCD, and that failures in cognitive and behavioural inhibitory processes appear to underlie many of the symptoms and neuroc cognitive findings.
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Effects of lesions to ascending noradrenergic neurones on performance of a 5-choice serial reaction task in rats; implications for theories of dorsal noradrenergic bundle function based on selective attention and arousal
TL;DR: Although intense white noise failed to produce differential impairments when presented simultaneously with the visual discriminanda, the DNAB lesion significantly impaired accuracy when the noise was presented immediately prior to, but not overlapping, the onset of the visual stimuli.
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Defining the neural mechanisms of probabilistic reversal learning using event-related functional magnetic resonance imaging.
TL;DR: Event-related functional magnetic resonance imaging was used to measure blood oxygenation level-dependent responses in 13 young healthy human volunteers during performance of a probabilistic reversal-learning task, finding that signal change in this ventral frontostriatal circuit is associated with reversal learning and is uncontaminated by negative feedback.