Y
Yu Huang
Researcher at Chinese Academy of Sciences
Publications - 1798
Citations - 114451
Yu Huang is an academic researcher from Chinese Academy of Sciences. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 136, co-authored 1492 publications receiving 89209 citations. Previous affiliations of Yu Huang include The Chinese University of Hong Kong & Samsung.
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Journal ArticleDOI
Blueberry anthocyanins at doses of 0.5 and 1 % lowered plasma cholesterol by increasing fecal excretion of acidic and neutral sterols in hamsters fed a cholesterol-enriched diet
TL;DR: The cholesterol-lowering activity of blueberry anthocyanins was most likely mediated by enhancing the excretion of sterols accompanied with down-regulation on gene expression of intestinal NPC1L1, ACAT-2, MTP, and ABCG 8.
Journal ArticleDOI
A Rational Biomimetic Approach to Structure Defect Generation in Colloidal Nanocrystals
Lingyan Ruan,Hadi Ramezani-Dakhel,Chain Lee,Yongjia Li,Xiangfeng Duan,Hendrik Heinz,Yu Huang +6 more
TL;DR: Mechanistic investigations of high density twin formation induced by specific peptides in platinum (Pt) NC growth are reported, on the basis of which principles that can serve as guidelines for the rational design of molecular surfactants to introduce high yield twinning in noble metal NC syntheses are derived.
Proceedings ArticleDOI
Effects of Embedded Decompression and Compaction Architectures on Side-Channel Attack Resistance
Chunsheng Liu,Yu Huang +1 more
TL;DR: The authors have shown that successful attacking of designs with embedded decompressor and compactor is extremely difficult and indicates that the use of embedded compression architectures can achieve higher security level.
Journal ArticleDOI
Urocortin-induced relaxation in the human internal mammary artery.
TL;DR: The vascular effect of urocortin on human internal mammary artery (IMA) in vitro and the possible underlying mechanisms are investigated to produce both endothelium-dependent and -independent relaxation in human IMA rings.
Journal ArticleDOI
Protein kinase C can inhibit TRPC3 channels indirectly via stimulating protein kinase G
TL;DR: Data strongly suggest that the inhibitory action of PKC on TRPC3 is partly mediated through PKG in these PKG‐overexpressing cells, and an important role of PKG is suggested in the PMA‐induced inhibition of TRPC channels in native endothelial cells.