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Yu Huang

Researcher at Chinese Academy of Sciences

Publications -  1798
Citations -  114451

Yu Huang is an academic researcher from Chinese Academy of Sciences. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 136, co-authored 1492 publications receiving 89209 citations. Previous affiliations of Yu Huang include The Chinese University of Hong Kong & Samsung.

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Proceedings ArticleDOI

On designing two-dimensional scan architecture for test chips

TL;DR: The concept of a two-dimensional scan architecture is presented that makes testing and diagnose down to defective scan cells more easily, and will help manufacture to incorporate silicon-accurate models and enhanced the manufacturing process.
Journal ArticleDOI

Whole-Genome Sequencing of Chinese Yellow Catfish Provides a Valuable Genetic Resource for High-Throughput Identification of Toxin Genes

TL;DR: A high-quality genome assembly of Chinese yellow catfish is assembled and high-throughput identification of toxin genes from a genomic view is performed, ensuring the limited number of toxin sequences in public databases will be remarkably improved once the authors integrate multi-omics data from more and more sequenced species.
Journal ArticleDOI

Experimental study on the stability of the ClHgSO3- in desulfurization wastewater.

TL;DR: It is proposed that the ClHgSO3− probably decomposes and releases Hg0 in two pathways, and changes of the water’s features could disturb the balance of Hg2+–Cl−–SO32− systems, which might stimulate the decomposition of ClHGSO3.
Journal ArticleDOI

Effect of hydrogen peroxide and superoxide anions on cytosolic Ca2+: comparison of endothelial cells from large-sized and small-sized arteries.

TL;DR: H2O2 could induce store Ca2+ release via phospholipase C-IP3 pathway in endothelial cells through the following pathways: superoxide anions, HX-XO, and Hypoxanthine-xanthine oxidase.
Journal ArticleDOI

A GLP-1 analog lowers ER stress and enhances protein folding to ameliorate homocysteine-induced endothelial dysfunction

TL;DR: This paper showed that exendin-4 reduces hyperhomocysteinemia (HHcy) induced endoplasmic reticulum (ER) stress and oxidative stress to impair endothelial function.