scispace - formally typeset
M

Monika Stoll

Researcher at Maastricht University

Publications -  155
Citations -  10973

Monika Stoll is an academic researcher from Maastricht University. The author has contributed to research in topics: Medicine & Genome-wide association study. The author has an hindex of 38, co-authored 122 publications receiving 9874 citations. Previous affiliations of Monika Stoll include University of Münster.

Papers
More filters
Journal ArticleDOI

Plasma HDL cholesterol and risk of myocardial infarction: A mendelian randomisation study

Benjamin F. Voight, +140 more
TL;DR: In this paper, a Mendelian randomisation analysis was performed to compare the effect of HDL cholesterol, LDL cholesterol, and genetic score on risk of myocardial infarction.
Journal ArticleDOI

Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease

Heribert Schunkert, +166 more
- 01 Apr 2011 - 
TL;DR: This paper performed a meta-analysis of 14 genome-wide association studies of coronary artery disease (CAD) comprising 22,233 individuals with CAD (cases) and 64,762 controls of European descent followed by genotyping of top association signals in 56,682 additional individuals.

Plasma HDL cholesterol and risk of myocardial infarction: a mendelian randomisation study

Benjamin F. Voight, +125 more
TL;DR: Mendelian randomisation analyses challenge the concept that raising of plasma HDL cholesterol will uniformly translate into reductions in risk of myocardial infarction.
Journal ArticleDOI

Genome-wide association of early-onset myocardial infarction with single nucleotide polymorphisms and copy number variants.

Sekar Kathiresan, +118 more
- 08 Feb 2009 - 
TL;DR: SNPs at nine loci were reproducibly associated with myocardial infarction, but tests of common and rare CNVs failed to identify additional associations with my Cardiovascular Infarction risk.
Journal ArticleDOI

Genetic variation in DLG5 is associated with inflammatory bowel disease

TL;DR: A significant difference in association of the 113A DLG5 variant with Crohn disease is found in affected individuals carrying the risk- associated CARD15 alleles versus those carrying non-risk-associated CARD 15 alleles, suggestive of a complex pattern of gene-gene interaction betweenDLG5 and CARD15, reflecting the complex nature of polygenic diseases.