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Mary Susan Burnett

Researcher at MedStar Washington Hospital Center

Publications -  67
Citations -  12855

Mary Susan Burnett is an academic researcher from MedStar Washington Hospital Center. The author has contributed to research in topics: Genome-wide association study & Angiogenesis. The author has an hindex of 35, co-authored 67 publications receiving 11548 citations. Previous affiliations of Mary Susan Burnett include MedStar Health & Memorial Hospital of South Bend.

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Discovery and refinement of loci associated with lipid levels

Cristen J. Willer, +319 more
- 06 Oct 2013 - 
TL;DR: It is found that loci associated with blood lipid levels are often associated with cardiovascular and metabolic traits, including coronary artery disease, type 2 diabetes, blood pressure, waist-hip ratio and body mass index.
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Plasma HDL cholesterol and risk of myocardial infarction: A mendelian randomisation study

Benjamin F. Voight, +140 more
TL;DR: In this paper, a Mendelian randomisation analysis was performed to compare the effect of HDL cholesterol, LDL cholesterol, and genetic score on risk of myocardial infarction.
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Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease

Heribert Schunkert, +166 more
- 01 Apr 2011 - 
TL;DR: This paper performed a meta-analysis of 14 genome-wide association studies of coronary artery disease (CAD) comprising 22,233 individuals with CAD (cases) and 64,762 controls of European descent followed by genotyping of top association signals in 56,682 additional individuals.
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Common variants associated with plasma triglycerides and risk for coronary artery disease

Ron Do, +266 more
- 01 Nov 2013 - 
TL;DR: It is suggested that triglyceride-rich lipoproteins causally influence risk for CAD, and the strength of a polymorphism's effect on triglyceride levels is correlated with the magnitude of its effect on CAD risk.
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Neuropeptide Y acts directly in the periphery on fat tissue and mediates stress-induced obesity and metabolic syndrome

TL;DR: It is reported that stress exaggerates diet-induced obesity through a peripheral mechanism in the abdominal white adipose tissue that is mediated by neuropeptide Y (NPY), and manipulations of NPY2R activity within fat tissue offer new ways to remodel fat and treat obesity and metabolic syndrome.