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Institution

Pusan National University

EducationBusan, South Korea
About: Pusan National University is a education organization based out in Busan, South Korea. It is known for research contribution in the topics: Catalysis & Population. The organization has 24124 authors who have published 45054 publications receiving 819356 citations. The organization is also known as: Busan National University & Pusan University.
Topics: Catalysis, Population, Thin film, Medicine, Apoptosis


Papers
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Journal ArticleDOI
TL;DR: A cost-efficient convolutional neural network for robust automatic modulation classification (AMC) deployed for cognitive radio services of modern communication systems and achieves the overall 24-modulation classification rate of 93.59% at 20 dB SNR on the well-known DeepSig dataset.
Abstract: This letter proposes a cost-efficient convolutional neural network (CNN) for robust automatic modulation classification (AMC) deployed for cognitive radio services of modern communication systems. The network architecture is designed with several specific convolutional blocks to concurrently learn the spatiotemporal signal correlations via different asymmetric convolution kernels. Additionally, these blocks are associated with skip connections to preserve more initially residual information at multi-scale feature maps and prevent the vanishing gradient problem. In the experiments, MCNet reaches the overall 24-modulation classification rate of 93.59% at 20 dB SNR on the well-known DeepSig dataset.

140 citations

Journal ArticleDOI
TL;DR: In this paper, the structural relationships between patrons' emotional responses induced by advertising, their perceived value, and their behavioral intentions in the chain restaurant industry were investigated, and which attributes of advertising bear the strongest impact on behavioral intentions.

140 citations

Journal ArticleDOI
TL;DR: The thermal, mechanical, and shape-memory effect of these TPUs of various block compositions and lengths were studied by differential scanning calorimetry, dynamic mechanical testing, and tensile testing as mentioned in this paper.
Abstract: Segmented thermoplastic polyurethanes (TPU)s with amorphous soft segments from the reaction of hexamethylene diisocyanate and 1,2-butanediol and crystalline hard segments from 4,4′-methylenediphenyl diisocyanate and 1,6-hexanediol showed sharp glass-transition temperatures that could be used as shape-recovery temperatures The thermal, mechanical, and shape-memory effect of these TPUs of various block compositions and lengths were studied by differential scanning calorimetry, dynamic mechanical testing, and tensile testing As the block lengths decreased, phase mixing increased and hysteresis in the shape-memory behavior decreased Too low a content of hard segments increased the hysteresis in the shape-memory behavior © 2000 John Wiley & Sons, Inc J Polym Sci B: Polym Phys 38: 2652–2657, 2000

140 citations

Journal ArticleDOI
TL;DR: It is suggested that salinomycin induces apoptosis, which was related to ROS-mediated autophagy through regulation of the PI3K/AKT/mTOR and ERK/p38 MAPK signaling pathways.
Abstract: Recently, the interplay between autophagy and apoptosis has become an important factor in chemotherapy for cancer treatment Inhibition of autophagy may be an effective strategy to improve the treatment of chemo-resistant cancer by consistent exposure to chemotherapeutic drugs However, no reports have clearly elucidated the underlying mechanisms Therefore, in this study, we assessed whether salinomycin, a promising anticancer drug, induces apoptosis and elucidated potential antitumor mechanisms in chemo-resistant prostate cancer cells Cell viability assay, Western blot, annexin V/propidium iodide assay, acridine orange (AO) staining, caspase-3 activity assay, reactive oxygen species (ROS) production, and mitochondrial membrane potential were assayed Our data showed that salinomycin alters the sensitivity of prostate cancer cells to autophagy Pretreatment with 3-methyladenine (3-MA), an autophagy inhibitor, enhanced the salinomycin-induced apoptosis Notably, salinomycin decreased phosphorylated of AKT and phosphorylated mammalian target of rapamycin (mTOR) in prostate cancer cells Pretreatment with LY294002, an autophagy and PI3K inhibitor, enhanced the salinomycin-induced apoptosis by decreasing the AKT and mTOR activities and suppressing autophagy However, pretreatment with PD98059 and SB203580, an extracellular signal-regulated kinases (ERK), and p38 inhibitors, suppressed the salinomycin-induced autophagy by reversing the upregulation of ERK and p38 In addition, pretreatment with N-acetyl-l-cysteine (NAC), an antioxidant, inhibited salinomycin-induced autophagy by suppressing ROS production Our results suggested that salinomycin induces apoptosis, which was related to ROS-mediated autophagy through regulation of the PI3K/AKT/mTOR and ERK/p38 MAPK signaling pathways

140 citations


Authors

Showing all 24296 results

NameH-indexPapersCitations
Hyun-Chul Kim1764076183227
Taeghwan Hyeon13956375814
George C. Schatz137115594910
Darwin J. Prockop12857687066
Mark A. Ratner12796868132
Csaba Szabó12395861791
David E. McClelland10760272881
Yong Sik Ok10285441532
C. M. Mow-Lowry10137866659
I. K. Yoo10143732681
Haijun Yang10040335114
Buddy D. Ratner9950135660
Dong Jo Kim9849736272
Shuzhi Sam Ge9788340865
B. J. J. Slagmolen9634962356
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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
202391
2022302
20213,260
20203,069
20193,039
20182,718