Institution
Tulane University
Education•New Orleans, Louisiana, United States•
About: Tulane University is a education organization based out in New Orleans, Louisiana, United States. It is known for research contribution in the topics: Population & Blood pressure. The organization has 24478 authors who have published 47205 publications receiving 1944993 citations. The organization is also known as: University of Louisiana.
Topics: Population, Blood pressure, Receptor, Poison control, Medicine
Papers published on a yearly basis
Papers
More filters
••
Emory University1, DePaul University2, University of Miami3, Baylor College of Medicine4, Pennsylvania State University5, University of California, Irvine6, Duke University7, University of Colorado Denver8, Medical University of South Carolina9, University of Oklahoma10, University of Texas Southwestern Medical Center11, University of Illinois at Chicago12, Tulane University13, University of California, San Francisco14, Icahn School of Medicine at Mount Sinai15, Harvard University16, Virginia Commonwealth University17, University of Pennsylvania18, Rush University Medical Center19, Oregon Health & Science University20
TL;DR: This document represents a continuation of the National Lipid Association recommendations developed by a diverse panel of experts who examined the evidence base and provided recommendations regarding the following topics: lifestyle therapies and strategies to improve patient outcomes by increasing adherence and using team-based collaborative care.
690 citations
••
TL;DR: An analytic representation of g\ifmmode\bar\else\textasciimacron\fi{} (and hence g) in real space for a uniform electron gas with density parameter ${\mathit{r}$ and spin polarization \ensuremath{\zeta}.
Abstract: The pair-distribution function g describes physical correlations between electrons, while its average g\ifmmode\bar\else\textasciimacron\fi{} over coupling constant generates the exchange-correlation energy. The former is found from the latter by g=(1-${\mathit{a}}_{0}$\ensuremath{\partial}/\ensuremath{\partial}${\mathit{a}}_{0}$)g\ifmmode\bar\else\textasciimacron\fi{}, where ${\mathit{a}}_{0}$ is the Bohr radius. We present an analytic representation of g\ifmmode\bar\else\textasciimacron\fi{} (and hence g) in real space for a uniform electron gas with density parameter ${\mathit{r}}_{\mathit{s}}$ and spin polarization \ensuremath{\zeta}. This expression has the following attractive features: (1) The exchange-only contribution is treated exactly, apart from oscillations we prefer to ignore. (2) The correlation contribution is correct in the high-density (${\mathit{r}}_{\mathit{s}}$\ensuremath{\rightarrow}0) and nonoscillatory long-range (R\ensuremath{\rightarrow}\ensuremath{\infty}) limits. (3) The value and cusp are properly described in the short-range (R\ensuremath{\rightarrow}0) limit. (4) The normalization and energy integrals are respected. The result is found to agree with the pair-distribution function g from Ceperley's quantum Monte Carlo calculation. Estimates are also given for the separate contributions from parallel and antiparallel spin correlations, and for the long-range oscillations at a high finite density.
689 citations
••
TL;DR: Waist circumference, which is relatively easy to measure, may help to identify children likely to have adverse concentrations of lipids and insulin.
689 citations
••
15 Nov 1984TL;DR: In this paper, an initial wave function or quantum density operator is expanded in a complete set of grussian wavepackets and it is demonstrated that the time evolution of this wavepacket expansion for the quantum wavefunction or density is correctly given within the approximations employed by the classical propagation of the avarage position and momentum of each gaussian packet, holding the shape of these individual gaussians fixed.
Abstract: A justification is given for the use of non-spreading or frozen gaussian packets in dynamics calculations In this work an initial wavefunction or quantum density operator is expanded in a complete set of grussian wavepackets It is demonstrated that the time evolution of this wavepacket expansion for the quantum wavefunction or density is correctly given within the approximations employed by the classical propagation of the avarage position and momentum of each gaussian packet, holding the shape of these individual gaussians fixed The semiclassical approximation is employed for the quantum propagator and the stationary phase approximation for certain integrals is utilized in this derivation This analysis demonstrates that the divergence of the classical trajectories associated with the individual gaussian packets accounts for the changes in shape of the quantum wavefunction or density, as has been suggested on intuitive grounds by Heller The method should be exact for quadratic potentials and this is verified by explicitly applying it for the harmonic oscillator example
686 citations
••
National Institutes of Health1, Conservatoire national des arts et métiers2, Harvard University3, Science Applications International Corporation4, Oakland University5, Marshfield Clinic6, Rush University Medical Center7, Yeshiva University8, University of Texas Medical Branch9, University of Pennsylvania10, Medical University of Białystok11, Tulane University12, Case Western Reserve University13, MetroHealth14, Johns Hopkins University15
TL;DR: African Americans carrying two APOL1 risk alleles have a greatly increased risk for glomerular disease, andAPOL1-associated FSGS occurs earlier and progresses to ESRD more rapidly, adding to the evidence base required to determine whether genetic testing for APol1 has a use in clinical practice.
Abstract: Trypanolytic variants in APOL1, which encodes apolipoprotein L1, associate with kidney disease in African Americans, but whether APOL1-associated glomerular disease has a distinct clinical phenotype is unknown. Here we determined APOL1 genotypes for 271 African American cases, 168 European American cases, and 939 control subjects. In a recessive model, APOL1 variants conferred seventeenfold higher odds (95% CI 11 to 26) for focal segmental glomerulosclerosis (FSGS) and twenty-ninefold higher odds (95% CI 13 to 68) for HIV-associated nephropathy (HIVAN). FSGS associated with two APOL1 risk alleles associated with earlier age of onset (P 0.01) and faster progression to ESRD (P 0.01) but similar sensitivity to steroids compared with other subjects. Individuals with two APOL1 risk alleles have an estimated 4% lifetime risk for developing FSGS, and untreated HIVinfected individuals have a 50% risk for developing HIVAN. The effect of carrying two APOL1 risk alleles explains 18% of FSGS and 35% of HIVAN; alternatively, eliminating this effect would reduce FSGS and HIVAN by 67%. A survey of world populations indicated that the APOL1 kidney risk alleles are present only on African chromosomes. In summary, African Americans carrying two APOL1 risk alleles have a greatly increased risk for glomerular disease, and APOL1-associated FSGS occurs earlier and progresses to ESRD more rapidly. These data add to the evidence base required to determine whether genetic testing for APOL1 has a use in clinical practice.
684 citations
Authors
Showing all 24722 results
Name | H-index | Papers | Citations |
---|---|---|---|
Walter C. Willett | 334 | 2399 | 413322 |
JoAnn E. Manson | 270 | 1819 | 258509 |
Frank B. Hu | 250 | 1675 | 253464 |
Eric B. Rimm | 196 | 988 | 147119 |
Krzysztof Matyjaszewski | 169 | 1431 | 128585 |
Nicholas J. White | 161 | 1352 | 104539 |
Tien Yin Wong | 160 | 1880 | 131830 |
Tomas Hökfelt | 158 | 1033 | 95979 |
Thomas E. Starzl | 150 | 1625 | 91704 |
Geoffrey Burnstock | 141 | 1488 | 99525 |
Joseph Sodroski | 138 | 542 | 77070 |
Glenn M. Chertow | 128 | 764 | 82401 |
Darwin J. Prockop | 128 | 576 | 87066 |
Kenneth J. Pienta | 127 | 671 | 64531 |
Charles Taylor | 126 | 741 | 77626 |