R
Roberto Romero
Researcher at National Institutes of Health
Publications - 1622
Citations - 121818
Roberto Romero is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Amniotic fluid & Pregnancy. The author has an hindex of 151, co-authored 1516 publications receiving 108321 citations. Previous affiliations of Roberto Romero include University of Michigan & Weizmann Institute of Science.
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Journal ArticleDOI
Prenatal diagnoses in unilateral empty renal fossa.
TL;DR: It is demonstrated that, in spite of their nonvital in utero role, single kidneys may show compensatory hypertrophy.
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Prenatal diagnosis of fetal cutaneous hemangioma: case report and review of the literature.
TL;DR: Hemangiomas are benign vascular tumors that represent the most common of all congenital anomalies and may involve skeletal muscle, heart, liver, bone, or central nervous system.
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Maternal plasma-soluble ST2 concentrations are elevated prior to the development of early and late onset preeclampsia - a longitudinal study.
Roberto Romero,Piya Chaemsaithong,Adi L. Tarca,Steven J. Korzeniewski,Eli Maymon,Percy Pacora,Bogdan Panaitescu,Noppadol Chaiyasit,Zhong Dong,Offer Erez,Sonia S. Hassan,Tinnakorn Chaiworapongsa +11 more
TL;DR: Maternal plasma sST2 concentrations are elevated 6 weeks prior to the clinical diagnosis of preeclampsia, which may contribute to an exaggerated intravascular inflammatory response and/or the Th1/Th2 imbalance in some cases.
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Fetal growth cessation in late pregnancy: its impact on predicted size parameters used to classify small for gestational age neonates.
TL;DR: Growth cessation occurred at approximately 38 weeks for all six birth characteristics studied, and SGA neonates can be separated into normal and growth restricted subgroups based on the frequency of abnormal GPRI values (GPRI Profile Classification).
Failure of physiologic transformation of spiral arteries, endothelial and trophoblast cell activation, and acute atherosis in the basal plate of the placenta
Carlos A. Labarrere,Hector L. DiCarlo,Elaine Bammerlin,James W. Hardin,Yeon Mee Kim,Piya Chaemsaithong,David M. Haas,Ghassan S. Kassab,Roberto Romero +8 more
TL;DR: Failure of spiral arteries physiologic transformation in the placental basal plate is associated with interstitial extravillous trophoblast and arterial endothelial activation along with increased frequency of spiral artery atherosis.