The determination of ascorbic acid in whole blood and urine through the 2,4-dinitrophenylhydrazine derivative of dehydroascorbic acid
Joseph H. Roe,Carl A. Kuether +1 more
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This article is published in Journal of Biological Chemistry.The article was published on 1943-02-01 and is currently open access. It has received 1689 citations till now. The article focuses on the topics: Ascorbic acid & Dehydroascorbic acid.read more
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Biochemical Changes in Organs of Chicks and Rats Poisoned with Ethylene Dibromide and Carbon Tetrachloride
TL;DR: Rats and chicks responded differently to toxic action of the two poisons, which may be related to the different metabolism of the poisons in the two species.
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Polarographic Investigations of Vitamin C. III. Reduction Waves of the Condensation Products of Dehydro-L-ascorbic Acid and its Related Compounds with o-Phenylenediamine. Application to the Simultaneous Determination of L-Ascorbic Acid, Dehydro-L-ascorbic Acid and their Related Compounds
TL;DR: In this article, the polarographic behavior of dehydro-L-ascorbic acid (DAA), dehydroreductic acid, DRA, alloxan, mesoxalaldehyde (MOA), and 2,3-diketogulonic acid in acid buffer solutions containing o-phenylenediamine (OPD), has been investigated.
Vitamin C in the Diet of Inuit Hunters From Holman, Northwest Territories
Joseph R. Geraci,Thomas G. Smith +1 more
TL;DR: In this article, during the spring and summer months the diet of three Inuit families living in a seal hunting camp south of Holman, N.W.T., was studied.
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Asiatic acid attenuates pre-neoplastic lesions, oxidative stress, biotransforming enzymes and histopathological alterations in 1,2-dimethylhydrazine-induced experimental rat colon carcinogenesis.
Aktarul Islam Siddique,Vijay Mani,Sivaranjani Arivalagan,Nisha Susan Thomas,Nalini Namasivayam +4 more
TL;DR: The article is being retracted due to image manipulation in Figures 3 and 9, and the authors were not able to provide original data that would support the integrity of their research.
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Effects of chronic ethanol consumption in blood: A time dependent study on rat.
TL;DR: It is concluded that long-term ethanol-induced toxicity is linked to an oxidative stress, which may aggravate to fibrosis and elevate pro-angiogenic factors, but not associated with lymphangiogenesis.
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