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Open AccessJournal ArticleDOI

The determination of ascorbic acid in whole blood and urine through the 2,4-dinitrophenylhydrazine derivative of dehydroascorbic acid

Joseph H. Roe, +1 more
- 01 Feb 1943 - 
- Vol. 147, Iss: 2, pp 399-407
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This article is published in Journal of Biological Chemistry.The article was published on 1943-02-01 and is currently open access. It has received 1689 citations till now. The article focuses on the topics: Ascorbic acid & Dehydroascorbic acid.

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Effect of chrysin on hepatoprotective and antioxidant status in d-galactosamine-induced hepatitis in rats

TL;DR: It is demonstrated that chrysin acts as a hepatoprotective and antioxidant agent against d-galactosamine-induced hepatotoxicity and reduces glutathione, vitamin C and vitamin E.
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Differential rapid analysis of ascorbic acid and ascorbic acid 2-sulfate by dinitrophenylhydrazine method.

TL;DR: The differential oxidation of AA and AAS with 2, 6-dinitrophenolindophenol (2,6-Dye) and KBrO 3 and the determination of the osazone produced with the original method by Roe and Kuether are concerned.
Journal ArticleDOI

Respiration and glucose oxidation in human and guinea pig leukocytes: comparative studies

TL;DR: A comparison has been made of the metabolic shifts in human and guinea pig leukocytes when they phagocytize and enzymes potentially responsible for these phenomena have been compared in each species.
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Selenium modulates dynamics of antioxidative defence expression, photosynthetic attributes and secondary metabolites to mitigate chromium toxicity in Brassica juncea L. plants

TL;DR: In this paper, the stress protective role of selenium (Se) against chromium toxicity in Brassica juncea L. has been investigated and shown that Se is able to protect the plants by restoring growth, reducing the oxidative damage (low levels of superoxide anions and hydrogen peroxide), and strengthened the enzymatic and nonenzymatic defence systems of B.juncea plants.
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Protective effects of dietary cyanidin 3-O-β-D-glucoside on liver ischemia-reperfusion injury in rats

TL;DR: This study evaluated whether feeding C3G suppresses oxidative injury to the liver caused by hepatic ischemia-reperfusion (I/R), which was used as a model for oxidative stress and indicates that orally administered C3g suppresses I/R-induced oxidative damage and suggests that C2G functions as a potent antioxidant in vivo under oxidative stress.
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