The determination of ascorbic acid in whole blood and urine through the 2,4-dinitrophenylhydrazine derivative of dehydroascorbic acid
Joseph H. Roe,Carl A. Kuether +1 more
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This article is published in Journal of Biological Chemistry.The article was published on 1943-02-01 and is currently open access. It has received 1689 citations till now. The article focuses on the topics: Ascorbic acid & Dehydroascorbic acid.read more
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Oxidative stress mitigation and initiation of antioxidant and osmoprotectant responses mediated by ascorbic acid in Brassica juncea L. subjected to copper (II) stress
Resham Sharma,Renu Bhardwaj,Ashwani Kumar Thukral,Asma A. Al-Huqail,Manzer H. Siddiqui,Parvaiz Ahmad,Parvaiz Ahmad +6 more
TL;DR: This study provides sufficient evidence regarding the protective role of ascorbic acid (AA) against ROS and its suggested use as a soil amendment against Cu (II) toxicity in B. juncea.
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Effects of umbelliferone on lipid peroxidation and antioxidant status in diethylnitrosamine-induced hepatocellular carcinoma
TL;DR: It is suggested that umbelliferone treatment prevents liver damage, lipid peroxidation and protects the antioxidant defense system in DEN-induced liver carcinogenesis in rats.
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Modulation of the Functional Components of Growth, Photosynthesis, and Anti-Oxidant Stress Markers in Cadmium Exposed Brassica juncea L.
Dhriti Kapoor,Mahendra Pratap Singh,Satwinderjeet Kaur,Renu Bhardwaj,Bingsong Zheng,Anket Sharma +5 more
TL;DR: It is suggested that Cd exposure to the seedlings triggered an induction of several defense responses in B. juncea including major metabolites, andMalondialdehyde and H2O2 contents were increased accompanied by enhanced Cd uptake.
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Oxidative stress induced by cadmium in the plasma, erythrocytes and lymphocytes of rats: Attenuation by grape seed proanthocyanidins
TL;DR: It is revealed that GSP affords superior protection against Cd-induced reactive oxygen species generation, lipid peroxidation, and free radical generation in C d-treated rats, which presumably reflects the ability of this flavonoid to protect erythrocytes and lymphocytes of rats from the toxic effects of Cd.
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Ascorbic acid deficiency and induction of hepatic microsomal hydroxylative enzymes by organochlorine pesticides.
D.J. Wagstaff,Joseph C. Street +1 more
TL;DR: Maintenance of induction was related to dietary levels rather than hepatic concentrations of the vitamin and required higher dietary levels of ascorbic acid than scurvy prevention, and sex had no effect on either induction by dieldrin or its impairment in ascorBic acid deficiency.
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