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He Li

Researcher at Beijing Technology and Business University

Publications -  59
Citations -  549

He Li is an academic researcher from Beijing Technology and Business University. The author has contributed to research in topics: Chemistry & Medicine. The author has an hindex of 8, co-authored 27 publications receiving 262 citations. Previous affiliations of He Li include University of Virginia & South China Normal University.

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Measurements of properties of the Higgs boson decaying to a W boson pair in pp collisions at s=13TeV

Albert M. Sirunyan, +2296 more
- 10 Apr 2019 - 
TL;DR: The first observation of the Higgs boson decay to W boson pairs by the LHC experiment was reported in this article, where the cross section times branching fraction was 1.28−0.17+0.18 times the standard model prediction.
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Measurements of tt¯differential cross sections in proton-proton collisions at √s=13 TeV using events containing two leptons

Albert M. Sirunyan, +2360 more
TL;DR: In this article, measurements of differential top quark pair cross sections using events produced in proton-proton collisions at a centre-of-mass energy of 13 TeV containing two oppositely charged leptons are presented.
Journal ArticleDOI

Measurement of the mass difference between neutral charm-meson eigenstates

Roel Aaij, +843 more
TL;DR: These determinations are the most precise from a single experiment and, combined with current world-average results, yield the first evidence that the masses of the neutral charm-meson eigenstates differ.
Journal ArticleDOI

Measurement of differential cross sections for Z boson pair production in association with jets at s=8 and 13 TeV

Albert M. Sirunyan, +2301 more
- 10 Feb 2019 - 
TL;DR: In this paper, the authors reported measurements of differential cross sections for the production of two Z bosons in association with jets in proton-proton collisions at root s = 8 and 13 TeV.
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Soybean protein-derived peptides inhibit inflammation in LPS-induced RAW264.7 macrophages via the suppression of TLR4-mediated MAPK-JNK and NF-kappa B activation.

TL;DR: The findings showed that SBP impeded the increase of toll-like receptor 4 activity by restricting LY96, while also inhibiting the mitogen-activated protein kinase-c-Jun N-terminal kinase pathway in cells, preventing LPS-induced inflammation of RAW 264.7 cells.