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Zhaoming Wang

Researcher at National Institutes of Health

Publications -  26
Citations -  1599

Zhaoming Wang is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Genome-wide association study & Single-nucleotide polymorphism. The author has an hindex of 19, co-authored 26 publications receiving 1348 citations. Previous affiliations of Zhaoming Wang include St. Jude Children's Research Hospital & Nanjing Medical University.

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Genome-wide association study of prostate cancer in men of African ancestry identifies a susceptibility locus at 17q21

Christopher A. Haiman, +62 more
- 01 Jun 2011 - 
TL;DR: A new risk variant on chromosome 17q21 is identified, ∼5% in men of African descent, whereas it is rare in other populations (<1%).

Genome-wide association study identifies multiple susceptibility loci for diffuse large B cell lymphoma

James R. Cerhan, +132 more
TL;DR: This paper conducted a meta-analysis of three new genome-wide association studies (GWAS) and 1 previous scan, totaling 3,857 cases and 7,666 controls of European ancestry, with additional genotyping of 9 promising SNPs in 1,359 cases and 4,557 controls.
Journal ArticleDOI

Genome-wide association study identifies multiple susceptibility loci for diffuse large B cell lymphoma.

James R. Cerhan, +132 more
- 01 Nov 2014 - 
TL;DR: Data provide substantial new evidence for genetic susceptibility to this B cell malignancy and point to pathways involved in immune recognition and immune function in the pathogenesis of DLBCL.
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Genome-wide association study identifies multiple risk loci for renal cell carcinoma.

Ghislaine Scelo, +129 more
TL;DR: This paper conducted a meta-analysis of two new scans of 5,198 cases and 7,331 controls together with four existing scans, totalling 10,784 cases and 20,406 controls of European ancestry.
Journal ArticleDOI

Two susceptibility loci identified for prostate cancer aggressiveness

TL;DR: A multistage, case-only genome-wide association study of prostate cancer cases identifies two loci associated with Gleason score, a pathological measure of disease aggressiveness, and the proximity of these loci to genes involved in vascular disease suggests potential biological mechanisms worthy of further investigation.