Institution
Fu Jen Catholic University
Education•Taipei, Taiwan•
About: Fu Jen Catholic University is a education organization based out in Taipei, Taiwan. It is known for research contribution in the topics: Population & Medicine. The organization has 6842 authors who have published 9512 publications receiving 171005 citations. The organization is also known as: FJU & Fu Jen.
Topics: Population, Medicine, Cancer, Hazard ratio, Apoptosis
Papers published on a yearly basis
Papers
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TL;DR: The effect of different diagnostic definitions on both the population prevalence of diabetes and the classification of previously undiagnosed individuals as having diabetes versus not having diabetes in a pooled analysis of data from population-based health examination surveys in different regions is assessed.
126 citations
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TL;DR: Most of the research about the hypoglycemic action effects of polysaccharides, proteoglycans, proteins and tritrerpenoids from G. lucidum are summarized to serve as a guide for future research.
125 citations
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TL;DR: It is hypothesized that the inhibitory mechanisms of emodin on activated T cells proliferation are related to the impairment of cytokine production, IL-2 mRNA level and [Ca2+]i in the cells.
Abstract: Objective and Design: This study was designed to elucidate action mechanisms of four anthraquinones identified from Polygonum hypoleucum Ohwi (P. hypoleucum Ohwi) on primary human T lymphocytes.¶Material and methods: The cells were isolated from peripheral blood.¶Treatment: T cells were treated with 5 to 60 μM of four anthraquinones with or without phytohemagglutinin (PHA; 5 μg/ml) for 3 days. Effects of 4 anthraquinones on T lymphocyte proliferation, production and gene expression of inflammatory cytokines and intracellular free Ca2+ concentration ([Ca2+]i) were determined. Data were assessed with Student's t-test.¶Results: On a percentage basis, emodin had the highest suppressing activity on T lymphocyte proliferation with an IC50 of 11.2 ± 0.6 μM. Emodin decreased cytokine production, IL-2 mRNA expression, and [Ca2+]i in activated T cells.¶Conclusions: We hypothesize that the inhibitory mechanisms of emodin on activated T cells proliferation are related to the impairment of cytokine production, IL-2 mRNA level and [Ca2+]i in the cells.¶
125 citations
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University of Tokyo1, Budker Institute of Nuclear Physics2, Novosibirsk State University3, École Polytechnique Fédérale de Lausanne4, University of Sydney5, University of Melbourne6, Panjab University, Chandigarh7, Polish Academy of Sciences8, University of Maribor9, Fu Jen Catholic University10, National Central University11, Hanyang University12, National Taiwan University13, Yonsei University14, Gyeongsang National University15, Sungkyunkwan University16, University of Cincinnati17, Tokyo Institute of Technology18, University of Ljubljana19, Korea University20, Shinshu University21, Nagoya University22, Tohoku Gakuin University23, Kyungpook National University24, Saga University25, Okayama University26, Tata Institute of Fundamental Research27, Chiba University28, Niigata University29, Graduate University for Advanced Studies30, University of Giessen31, Seoul National University32, University of Science and Technology of China33, Princeton University34, Nara Women's University35, Osaka City University36, Tokyo University of Agriculture and Technology37, Kanagawa University38, Virginia Tech39, Austrian Academy of Sciences40, University of Nova Gorica41, Tokyo Metropolitan University42, National United University43
TL;DR: In this paper, a Dalitz plot analysis of B→Kπ+ψ′ decays was performed on a 605fb-1 data sample that contains 657×106 BB pairs collected near the Υ(4S) resonance with the Belle detector at the KEKB asymmetric energy e+e-collider.
Abstract: From a Dalitz plot analysis of B→Kπ+ψ′ decays, we find a signal for Z(4430)+→π+ψ′ with a mass M=(4443-12-13+15+19)MeV/ c2, width Γ=(107-43-56+86+74)MeV, product branching fraction B(B0→K-Z(4430)+)×B(Z(4430)+→π+ψ′)=(3.2-0.9-1. 6+1.8+5.3)×10-5, and significance of 6.4σ that agrees with previous Belle measurements based on the same data sample. In addition, we determine the branching fraction B(B0→K*(892)0ψ′)=(5.52-0.32-0.58+0.35+0. 53)×10-4 and the fraction of K*(892)0 mesons that are longitudinally polarized fL=(44.8-2.7-5.3+4.0+4.0)%. These results are obtained from a 605fb-1 data sample that contains 657×106 BB pairs collected near the Υ(4S) resonance with the Belle detector at the KEKB asymmetric-energy e+e- collider. © 2009 The American Physical Society.
125 citations
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TL;DR: Compound 10 showed a significant inhibitory effect on the release of beta-glucuronidase from rat neutrophils stimulated with formyl-Met-Leu-Phe (fMLP)/cytochalasin B (CB) whereas compound 9 significantly inhibited superoxide anion formation in fMLP/CB-stimulated rat neutrophic cells.
125 citations
Authors
Showing all 6861 results
Name | H-index | Papers | Citations |
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P. Chang | 170 | 2154 | 151783 |
Christian Guilleminault | 133 | 897 | 68844 |
Pan-Chyr Yang | 102 | 786 | 46731 |
Po-Ren Hsueh | 92 | 1030 | 38811 |
Shyi-Ming Chen | 90 | 425 | 22172 |
Peter J. Rossky | 74 | 280 | 21183 |
Chong-Jen Yu | 72 | 577 | 22940 |
Shuu Jiun Wang | 71 | 502 | 24800 |
Jaw-Town Lin | 67 | 434 | 15482 |
Lung Chi Chen | 63 | 267 | 13929 |
Ronald E. Taam | 59 | 290 | 12383 |
Jiann T. Lin | 58 | 190 | 10801 |
Yueh-Hsiung Kuo | 57 | 618 | 12204 |
San Lin You | 55 | 178 | 16572 |
Liang-Gee Chen | 54 | 582 | 12073 |