Georgia State University

About: Georgia State University is a education organization based out in Atlanta, Georgia, United States. It is known for research contribution in the topics: Population & Poison control. The organization has 13988 authors who have published 35895 publications receiving 1164332 citations. The organization is also known as: GSU & Georgia State.

Current technological impediments to business-to-consumer electronic commerce

Abstract: Internet and World Wide Web technologies provide the infrastructure for the Electronic Commerce (e-Commerce) revolution now taking place. As a result of these technologies, even the smallest organization can afford to market its wares to hundreds of millions of potential e-Consumers. However, these technologies also pose threats to the very electronic commerce which they enable. For managers to strategize and implement e-Commerce effectively in their organizations, these impediments need to be recognized and understood. While hundreds of articles identify problems with Internet computing or conducting eCommerce, no unified framework of technological impediments specific to eCommerce yet exists.

A first study of the multidimensional students' life satisfaction scale with adolescents

Abstract: The psychometric properties of the MultidimensionalStudents’ Life Satisfaction Scale (MSLSS: Huebner, 1994) were assessed with a sample of adolescents fromgrades 9–12. Results of confirmatory factor analysessupported the hypothesized factor structure. Further,evidence for the convergent and discriminant validityof the instrument was obtained. Acceptable internalconsistency estimates for the Total and domain scoreswere also obtained. These psychometric propertieswere similar to findings of the MSLSS with youngerchildren. Limitations and future researchconsiderations were discussed.

Metformin Suppresses Diabetes-Accelerated Atherosclerosis via the Inhibition of Drp1-Mediated Mitochondrial Fission.

Abstract: Metformin is a widely used antidiabetic drug that exerts cardiovascular protective effects in patients with diabetes. How metformin protects against diabetes-related cardiovascular diseases remains poorly understood. Here, we show that metformin abated the progression of diabetes-accelerated atherosclerosis by inhibiting mitochondrial fission in endothelial cells. Metformin treatments markedly reduced mitochondrial fragmentation, mitigated mitochondrial-derived superoxide release, improved endothelial-dependent vasodilation, inhibited vascular inflammation, and suppressed atherosclerotic lesions in streptozotocin (STZ)-induced diabetic ApoE−/− mice. In high glucose–exposed endothelial cells, metformin treatment and adenoviral overexpression of constitutively active AMPK downregulated mitochondrial superoxide, lowered levels of dynamin-related protein (Drp1) and its translocation into mitochondria, and prevented mitochondrial fragmentation. In contrast, AMPK-α2 deficiency abolished the effects of metformin on Drp1 expression, oxidative stress, and atherosclerosis in diabetic ApoE−/−/AMPK-α2−/− mice, indicating that metformin exerts an antiatherosclerotic action in vivo via the AMPK-mediated blockage of Drp1-mediated mitochondrial fission. Consistently, mitochondrial division inhibitor 1, a potent and selective Drp1 inhibitor, reduced mitochondrial fragmentation, attenuated oxidative stress, ameliorated endothelial dysfunction, inhibited inflammation, and suppressed atherosclerosis in diabetic mice. These findings show that metformin attenuated the development of atherosclerosis by reducing Drp1-mediated mitochondrial fission in an AMPK-dependent manner. Suppression of mitochondrial fission may be a therapeutic approach for treating macrovascular complications in patients with diabetes.

The Mechanisms and Outcomes of Evaluation Influence

Abstract: Past literature has identified several putative precursors of use, as well as alternative forms of use. However, important shortcomings still exist in previous work on use. In particular, inadequate attention has been given to the underlying processes that may mediate the effects of evaluation on attitude and action. In essence, a key part of the theory of change for evaluation itself is missing. To help fill this gap, we describe a framework designed to capture key mechanisms through which evaluation may have its effects. The framework includes change processes that have been validated in various social science literatures. It identifies three levels of analysis (individual, interpersonal and collective), each with four kinds of processes (general influence, attitudinal, motivational and behavioral). With a more comprehensive view of the mechanisms underlying evaluation’s influence, the field can move forward in relation to its understanding and facilitation of evaluation’s role in the service of social ...

Are Some Emotions Marked "Whites Only"? Racialized Feeling Rules in Professional Workplaces

Abstract: Much of the research on emotion work in organizations has focused on the ways in which emotional per- formance reproduces gender inequality Yet, most of these studies overlook the racial character of professional workplaces and how emotion work is experienced by racial/ethnic minorities In this article, I examine how the normative feeling rules that guide emotional performance in professional workplaces are racialized rather than neutral or objective criteria Based on 25 semistructured interviews with black professionals, contend that feeling rules have different implications for black workers and ultimately reinforce racial difference This research contributes to the sociological literature on emotion work by further developing the racial components of emotional performance Keywords: feeling rules, emotion work, race, black professionals, tokenism