IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Humans
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This article is published in Journal of Clinical Pathology.The article was published on 1980-01-01 and is currently open access. It has received 3514 citations till now.read more
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A Compilation of Physical, Spectroscopic and Photophysical Properties of Polycyclic Aromatic Hydrocarbons
Reza Dabestani,Ilia N. Ivanov +1 more
TL;DR: A large body of work on this subject has appeared in the literature in recent years as discussed by the authors, including partitioning of polycyclic aromatic hydrocarbons (PAH) between air, water, soil or sediments.
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Modeling Smoking History: A Comparison of Different Approaches
TL;DR: The aim of this investigation was to elucidate the impact of several decisions that must be made when modeling smoking variables, using data on lung cancer from a case-control study undertaken in Montreal, Quebec, Canada, in 1979-1985.
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Internal exposure of the general population to DEHP and other phthalates--determination of secondary and primary phthalate monoester metabolites in urine.
TL;DR: The concentrations of the secondary, chain oxidized monoester metabolites of DEHP, mono(2-ethyl-5-hydroxyhexyl)phthalate (5OH-MEHP) and mono-n-octylphthalates (MEP) and 5oxo-ME HP are determined in urine samples from the general population to help to perform health risk assessments for the phthalate exposure of the generalpopulation.
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Environmental co-factors in HPV carcinogenesis.
TL;DR: It can be concluded that, among HPV positive women, high parity, long-term OC use, smoking, and co-infection with other sexually transmitted agents are the most consistently identified environmental co-factors likely to influence the risk of progression from cervical HPV infection to HSIL and invasive CC.
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Carcinogenicity and modification of the carcinogenic response by BHA, BHT, and other antioxidants.
TL;DR: Results indicate that BHA should be classified in the category of "sufficient evidence of carcinogenicity" as judged by IARC criteria, and the 3-tert isomer of BHA seemed to be responsible for the carcinogensicity of crude BHA in the forestomach of rats.