IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Humans
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This article is published in Journal of Clinical Pathology.The article was published on 1980-01-01 and is currently open access. It has received 3514 citations till now.read more
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Major Sources of Benzene Exposure
TL;DR: Data from EPA's TEAM Study allow us to identify the major sources of exposure to benzene for much of the U.S. population, and these sources turn out to be quite different from what had previously been considered the important sources.
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Acrylamide exposure from foods of the Dutch population and an assessment of the consequent risks
E.J.M Konings,A.J Baars,J.D. van Klaveren,M. C. Spanjer,P.M Rensen,M Hiemstra,J.A van Kooij,P.W.J Peters +7 more
TL;DR: It appears that the additional cancer risk from acrylamide exposure might not be negligible, and the public health risks of this consumption should be estimated.
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A clonal marker induced by mutation in mouse intestinal epithelium.
TL;DR: It is shown that this mutation-induced marker can be used to study the clonal organization of adult intestinal epithelium, and to mark descendent clones arising during development, and can in principle be extended to any other suitable markers which can be obtained in a heterozygous state.
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Effects of the phytochemicals, curcumin and quercetin, upon azoxymethane-induced colon cancer and 7,12-dimethylbenz[a]anthracene-induced mammary cancer in rats.
Michael A. Pereira,Clinton J. Grubbs,Leta H. Barnes,Hong Li,Greg R. Olson,Isao Eto,M. Margaret Juliana,Whitaker Lm,Gary J. Kelloff,Vernon E. Steele,Ronald A. Lubet +10 more
TL;DR: While curcumin was highly effective as a chemopreventive agent in the colon model, it was only weakly effective in the mammary model and quercetin caused a dose-dependent enhancement of tumors induced by AOM in the Colon model.
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Genetic polymorphism in the biotransformation of inorganic arsenic and its role in toxicity.
TL;DR: There seems to be a genetic polymorphism in the biomethylation of arsenic, however, the methyltransferases involved in arsenic methylation have not been characterized.