IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Humans
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This article is published in Journal of Clinical Pathology.The article was published on 1980-01-01 and is currently open access. It has received 3514 citations till now.read more
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Journal ArticleDOI
Aberrant p16 promoter methylation in smokers and former smokers with nonsmall cell lung cancer
Sonata Jarmalaite,Sonata Jarmalaite,Annamaria Kannio,Sisko Anttila,Juozas R. Lazutka,Kirsti Husgafvel-Pursiainen +5 more
TL;DR: Results suggest that analysis of promoter methylation in TSGs may provide a valuable biomarker for identification of groups with an elevated risk of cancer, such as smokers and ex‐smokers.
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Bitumen fumes: review of work on the potential risk to workers and the present knowledge on its origin
TL;DR: There is an interest in determining the chemical identity of the major DNA adducts induced by bitumen fume condensates to allow experimental studies on the carcinogenic potency of these compounds and their validation as potential biomarkers.
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Lymphohaematopoeitic cancer mortality among workers with benzene exposure
TL;DR: The observed number of deaths is small, and the number of peak exposures greater than 100 ppm to benzene is a better predictor of risk than cumulative exposure, which may be important factors for evaluating lymphohaematopoietic risk.
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Modelling of the individual and combined effects of water activity and temperature on the radial growth of Aspergillus flavus and A. parasiticus on corn.
Simbarashe Samapundo,Frank Devlieghere,A.H. Geeraerd,B. De Meulenaer,J.F. Van Impe,Johan Debevere +5 more
TL;DR: The Gibson and linear Arrhenius-Davey model describing the individual effects of a(w) or temperature on g or lambda proved an adequate predictor of either growth parameter and can be applied in the preservation of corn and the development of models that incorporate other factors important to mould growth on corn.
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Cell-mediated immunity and head and neck cancer: with special emphasis on betel quid chewing habit.
TL;DR: To elucidate how virus infection, tobacco, alcohol and BQ consumption, and other environmental exposure affect the immune status of patients with oral premalignant lesions or OC will help to understand the immunopathogenesis of OC and to develop immunotherapeutic strategies for OC.