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IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Humans

R. L. Carter
- 01 Jan 1980 - 
- Vol. 33, Iss: 1, pp 98-98
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This article is published in Journal of Clinical Pathology.The article was published on 1980-01-01 and is currently open access. It has received 3514 citations till now.

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Journal ArticleDOI

"IARC group 2B Carcinogens" reported in cigarette mainstream smoke.

TL;DR: Nine chemical compounds of the 50 Group 2A listings have been reported in cigarette mainstream smoke and in micrograms/cigarette (mug/cig), the ranges reported for each of the nine compounds are revealed.
Journal ArticleDOI

Carcinogenicity of metal compounds: possible role of DNA repair inhibition.

TL;DR: Current evidence suggests that DNA repair systems are very sensitive targets for nickel, cadmium, cobalt and arsenic, leading to a diminished removal of endogenous DNA lesions and of DNA damage induced by environmental agents, which in turn may increase the risk of tumor formation.
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Role of DNA repair inhibition in lead- and cadmium-induced genotoxicity: a review.

TL;DR: For both metals, the indirect genotoxic effects are observed at low, nontoxic concentrations, suggesting that an interference with DNA repair processes may be predominant at biologically relevant concentrations, and might explain the conflicting results of epidemiological studies obtained for both metals.
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Crohn's disease and cancer: A population-based cohort study

TL;DR: A population-based study of 1251 subjects with Crohn's disease diagnosed in Stockholm from 1955 to 1984 and followed in both the National Cancer Register and the National Cause-of-Death Register finds the occurrence of colorectal cancer was not increased.
Journal ArticleDOI

An analysis of risk factors for oral cancer in young people: a case-control study.

TL;DR: The results suggest that although this younger sample exhibit similar behavioural risk factors to older oral cancer patients, the low odds produced in addition to the relatively short duration of exposure, suggest that factors other than tobacco and alcohol may be implicated in the development of oral cancer in these younger patients.
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