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IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Humans

R. L. Carter
- 01 Jan 1980 - 
- Vol. 33, Iss: 1, pp 98-98
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This article is published in Journal of Clinical Pathology.The article was published on 1980-01-01 and is currently open access. It has received 3514 citations till now.

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Mechanisms of arsenic-induced cell transformation

TL;DR: The hypothesis that arsenic acts as a tumor progressor, rather than a tumor initiator or tumor promoter, is proposed, since amplification of oncogenes is observed in many human tumors.
Journal ArticleDOI

Lung cancer mortality in UK nickel-cadmium battery workers, 1947-2000.

TL;DR: The study findings do not support the hypotheses that cadmium compounds are human lung carcinogens, even when exposure histories were lagged first by 10, then by 20 years.
Journal ArticleDOI

Hematotoxicity and Carcinogenicity of Inhaled Benzene

TL;DR: Exposure of mice to 300 ppm for 2, 4, 8, and 16 weeks produced severe lymphopenia and decrease in marrow CFU-S, and inhalation of 3000 ppm for 8 days was less damaging than inhaling of 300 ppmFor 80 days (same integral amount of benzene inhaled).
Journal ArticleDOI

Toxicity of selected chlorobenzenes to aquatic organisms

TL;DR: In this article, an acute toxicity test with six chlorobenzenes was performed on several aquatic organisms at different trophic levels and the results were discussed together with physico-chemical properties of the molecules in order to identify structureactivity relationships and to predict environmental distribution.
Journal ArticleDOI

Diesel exhaust exposure and mortality among males in the American Cancer Society prospective study.

TL;DR: Railroad workers, heavy equipment operators, miners, and truck drivers had a higher mortality both for all causes and for lung cancer when compared with subjects with other occupations and no exposure to DE, and a dose-response effect was present.
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