IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Humans
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This article is published in Journal of Clinical Pathology.The article was published on 1980-01-01 and is currently open access. It has received 3514 citations till now.read more
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Exposure to dioxin and nonneoplastic mortality in the expanded IARC international cohort study of phenoxy herbicide and chlorophenol production workers and sprayers
John E. Vena,Paolo Boffetta,Heiko Becher,Trevor Benn,H B Bueno-de-Mesquita,David Coggon,Didier Colin,Dieter Flesch-Janys,Lois M. Green,Timo Kauppinen,Margareta Littorin,Elsebeth Lynge,John D. Mathews,Manfred Neuberger,Neil Pearce,Angela Cecilia Pesatori,R. Saracci,Kyle Steenland,Manolis Kogevinas +18 more
TL;DR: The authors studied noncancer mortality among phenoxyacid herbicide and chlorophenol production workers and sprayers included in an international study comprising 36 cohorts from 12 countries followed from 1939 to 1992 to suggest a moderate healthy worker effect for all circulatory diseases, especially ischemic heart disease.
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An IARC evaluation of polychlorinated dibenzo-p-dioxins and polychlorinated dibenzofurans as risk factors in human carcinogenesis.
TL;DR: The International Agency for Research on Cancer (IARC) Monographs program reevaluated polychlorinated dibenzo-p-dioxins and evaluated polychlorined dibenzofurans as possible carcinogenic hazards to humans in February 1997, using the most recent epidemiologic data on exposed human populations, experimental carcinogenicity bioassays in laboratory animals, and supporting evidence on relevant mechanisms of carcinogenesis.
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Acute, subacute and subchronic administration of methyl parathion-induced testicular damage in male rats and protective role of vitamins C and E
Meltem Uzunhisarcikli,Yusuf Kalender,Kerem Dirican,Suna Kalender,Ayse Ogutcu,Fatma Buyukkomurcu +5 more
TL;DR: In this paper, the effects of methyl parathion on the testes of rats were investigated at the end of 24 h, 4th and 7th weeks compared with a control group.
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Role of cobalt, iron, lead, manganese, mercury, platinum, selenium, and titanium in carcinogenesis.
TL;DR: The presently available data are inadequate to assess the possible carcinogenicity of selenium compounds, but a few observations suggest that selenIUM may suppress the effect of other carcinogens administered to experimental animals and may even be associated with lower cancer mortality rates in man.
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Multiple mechanisms for the carcinogenic effects of asbestos and other mineral fibers.
TL;DR: Although asbestos is inactive as a gene mutagen, there is now clear evidence that it induces chromosomal mutations (aneuploidy and aberrations) in a wide variety of mammalian cells including mesotheliomas and fibroblasts and a mechanism for cell transformation, which is dependent on fiber dimension, has been proposed.