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Shabnam Salimi

Researcher at University of Maryland, Baltimore

Publications -  39
Citations -  1133

Shabnam Salimi is an academic researcher from University of Maryland, Baltimore. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 15, co-authored 34 publications receiving 706 citations. Previous affiliations of Shabnam Salimi include Tehran University of Medical Sciences & Iran University of Medical Sciences.

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Measuring biological aging in humans: A quest.

TL;DR: Current research focuses on measuring the pace of aging to identify individuals who are “aging faster” to test and develop interventions that could prevent or delay the progression of multimorbidity and disability with aging.
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Assessing the contribution of rare variants to complex trait heritability from whole-genome sequence data

Pierrick Wainschtein, +453 more
TL;DR: The results imply that rare variants, in particular those in regions of low linkage disequilibrium, are a major source of the still missing heritability of complex traits and disease.
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Deep coverage whole genome sequences and plasma lipoprotein(a) in individuals of European and African ancestries.

Seyedeh M. Zekavat, +355 more
TL;DR: In this article, the authors used deep-coverage whole genome sequencing in 8392 individuals of European and African ancestry to discover and interpret both single-nucleotide variants and copy number (CN) variation associated with Lp(a).
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COVID-19 and Crosstalk With the Hallmarks of Aging.

TL;DR: Hallmarks of aging, interacting with one another, have been proposed to influence health span in older adults, possibly via mechanisms regulating the immune system, and these hallmarks likely contribute to the increased pathophysiological responses to SARS-CoV-2 among older adults.
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Microvascular endothelial dysfunction is associated with albuminuria and CKD in older adults

TL;DR: Microvascular endothelial function was strongly associated with greater albuminuria and CKD, independent of diabetes and blood pressure, and may explain in part the excess systemic cardiovascular risk associated with albuminurism and CKd.