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Wenge Wang

Researcher at University of Science and Technology of China

Publications -  200
Citations -  9264

Wenge Wang is an academic researcher from University of Science and Technology of China. The author has contributed to research in topics: Large Hadron Collider & Higgs boson. The author has an hindex of 42, co-authored 194 publications receiving 6992 citations. Previous affiliations of Wenge Wang include National University of Singapore & Osaka University.

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Journal ArticleDOI

An iron delivery pathway mediated by a lipocalin

Jun Yang, +10 more
- 01 Nov 2002 - 
TL;DR: It is shown that a member of the lipocalin superfamily (24p3/Ngal) delivers iron to the cytoplasm where it activates or represses iron-responsive genes and identifies an iron delivery pathway active in development and cell physiology.
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Search for electroweak production of charginos and sleptons decaying into final states with two leptons and missing transverse momentum in √s = 13 TeV pp collisions using the ATLAS detector

Georges Aad, +2937 more
- 14 Feb 2020 - 
TL;DR: In this article, a search for the electroweak production of charginos and sleptons decaying into final states with two electrons or muons is presented, based on 139.fb$^{-1}$ of proton-proton collisions recorded by the ATLAS detector at the Large Hadron Collider at
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The Myc–miR-17∼92 Axis Blunts TGFβ Signaling and Production of Multiple TGFβ-Dependent Antiangiogenic Factors

Michael Dews, +11 more
- 15 Oct 2010 - 
TL;DR: A pathway in which c-Myc activation of miR-17∼92 attenuates the TGFβ signaling pathway to shut down clusterin expression is defined, thereby stimulating angiogenesis and tumor cell growth.
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Reduction of TRAIL-Induced Mcl-1 and cIAP2 by c-Myc or Sorafenib Sensitizes Resistant Human Cancer Cells to TRAIL-Induced Death

M. Stacey Ricci, +12 more
- 10 Jul 2007 - 
TL;DR: It is found that c-Myc sensitized cells with defective intrinsic signaling to TRAIL and the combination of TRAIL with sorafenib holds promise for further development.
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Dual Inactivation of Akt and ERK by TIC10 Signals Foxo3a Nuclear Translocation, TRAIL Gene Induction, and Potent Antitumor Effects

Joshua E. Allen, +16 more
- 06 Feb 2013 - 
TL;DR: TIC10 is a small molecule that activates Foxo3a through dual inactivation of Akt and ERK, up-regulates the expression of the TRAIL gene, an endogenous tumor suppressor, and effectively improves the therapeutic properties and utility of TRAIL as an anticancer therapy.