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J. Ord

Researcher at St Bartholomew's Hospital

Publications -  6
Citations -  440

J. Ord is an academic researcher from St Bartholomew's Hospital. The author has contributed to research in topics: Antibody & Medicine. The author has an hindex of 4, co-authored 4 publications receiving 339 citations.

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Journal ArticleDOI

Use of leucocyte‐poor blood components and HLA‐matched‐platelet donors to prevent HLA alloimmunization

TL;DR: Improved methods of removing leucocytes from blood components appear to offer the best approach for minimizing HLA alloimmunization, as the provision of HLA‐matched platelet donors for prophylactic platelet support of all patients is not feasible.
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Baricitinib in patients admitted to hospital with COVID-19 (RECOVERY): a randomised, controlled, open-label, platform trial and updated meta-analysis

Obbina Abani, +7885 more
- 03 Mar 2022 - 
TL;DR: In patients hospitalised with COVID-19, baricitinib significantly reduced the risk of death but the size of benefit was somewhat smaller than that suggested by previous trials.
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Antenatal Management of Severe Feto-Maternal Alloimmune Thrombocytopenia: HLA Incompatibility May Affect Responses to Fetal Platelet Transfusions

TL;DR: This report describes a patient who lost three fetuses with ICH because of FMAIT due to anti-HPA-1a, the first time that HLA incompatibility has been found to complicate fetal transfusion therapy.
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Disappearance of HLA and platelet-specific antibodies in acute leukaemia patients alloimmunized by multiple transfusions.

TL;DR: Alloimmunization by platelet transfusions was studied in 154 patients with acute leukaemia and 37 patients with HLA antibodies including three with platelet‐specific antibodies and one patient with Platelet‐ specific antibodies alone survived their initial therapy and formed the basis of this study.
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Post-transfusion purpura associated with anti-Baka and anti-PIA2 platelet antibodies and delayed haemolytic transfusion reaction.

TL;DR: Clinically this was a typical case of PTP, but it was unusual serologically, and the patient's platelets were typed as homozygous PlA1‐positive and Baka‐negative.