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Andreas Pfeiffer

Researcher at CERN

Publications -  1920
Citations -  147114

Andreas Pfeiffer is an academic researcher from CERN. The author has contributed to research in topics: Large Hadron Collider & Lepton. The author has an hindex of 149, co-authored 1756 publications receiving 131080 citations. Previous affiliations of Andreas Pfeiffer include Heidelberg University & Paul Scherrer Institute.

Papers
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Search for dijet resonances in 7 TeV pp collisions at CMS

Vardan Khachatryan, +2199 more
TL;DR: In this article, a search for narrow resonances in the dijet mass spectrum is performed using data corresponding to an integrated luminosity of 2.9 inverse pb collected by the CMS experiment at the LHC.
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Search for Resonant Production of High-Mass Photon Pairs in Proton-Proton Collisions at s =8 and 13 TeV

Vardan Khachatryan, +2242 more
TL;DR: In this paper, a search for the resonant production of high-mass photon pairs is presented based on samples of proton-proton collision data collected by the CMS experiment at center-of-mass energies of 8 and 13 TeV, corresponding to integrated luminosities of 19.7 and 3.3 fb(-1).
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Search for dark matter produced with an energetic jet or a hadronically decaying W or Z boson at √s = 13 TeV

Albert M. Sirunyan, +2303 more
TL;DR: In this paper, a search for dark matter particles is performed using events with large missing transverse momentum, at least one energetic jet, and no leptons, in proton-proton collisions at root S = 13TeV collected with the CMS detector at the LHC.
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Interpretation of searches for supersymmetry with simplified models

S. Chatrchyan, +2170 more
- 23 Sep 2013 - 
TL;DR: In this article, the results of searches for supersymmetry by the CMS experiment are interpreted in the framework of simplified models, based on data corresponding to an integrated luminosity of 4.73 to 4.98 inverse femtobarns.
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Telomerase deficiency impairs glucose metabolism and insulin secretion.

TL;DR: In this article, the Terc subunit of the telomerase was found to exhibit impaired glucose tolerance in young adult mice, which is caused by impaired glucose-stimulated insulin secretion (GSIS) from pancreatic islets.