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Venkatesan Radha

Researcher at Indian Council of Medical Research

Publications -  85
Citations -  4095

Venkatesan Radha is an academic researcher from Indian Council of Medical Research. The author has contributed to research in topics: Population & Diabetes mellitus. The author has an hindex of 23, co-authored 66 publications receiving 3460 citations. Previous affiliations of Venkatesan Radha include Madras Medical College.

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Genome-wide trans-ancestry meta-analysis provides insight into the genetic architecture of type 2 diabetes susceptibility.

Anubha Mahajan, +395 more
- 01 Mar 2014 - 
TL;DR: In this paper, the authors aggregated published meta-analyses of genome-wide association studies (GWAS), including 26,488 cases and 83,964 controls of European, east Asian, south Asian and Mexican and Mexican American ancestry.
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Genome-wide association study in individuals of South Asian ancestry identifies six new type 2 diabetes susceptibility loci

Jaspal S. Kooner, +83 more
- 01 Oct 2011 - 
TL;DR: A genome-wide association study of type-2 diabetes in individuals of South Asian ancestry provides additional insight into mechanisms underlying T2D and shows the potential for new discovery from genetic association studies in South Asians.
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The GenomeAsia 100K Project enables genetic discoveries across Asia

TL;DR: The pilot phase of the GenomeAsia 100K Project catalogues genetic variation, population structure and disease associations to facilitate genetic studies in Asian populations and increase representation in genetics studies worldwide.
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ENPP1/PC-1 K121Q Polymorphism and Genetic Susceptibility to Type 2 Diabetes

TL;DR: The hypothesis that ENPP1 121Q predicts genetic susceptibility to type 2 diabetes in both South Asians and Caucasians is supports the hypothesis that the genotype-phenotype relationship is valid.
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Genome-Wide Association Study Identifies a Novel Locus Contributing to Type 2 Diabetes Susceptibility in Sikhs of Punjabi Origin From India

Richa Saxena, +56 more
- 01 May 2013 - 
TL;DR: The findings provide new evidence on the presence of a population-specific signal in relation to type 2 diabetes (T2D), which may provide additional insights into T2D pathogenesis.