Institution
American Cancer Society
Nonprofit•Atlanta, Georgia, United States•
About: American Cancer Society is a nonprofit organization based out in Atlanta, Georgia, United States. It is known for research contribution in the topics: Cancer & Population. The organization has 1339 authors who have published 3700 publications receiving 688166 citations. The organization is also known as: American Cancer Society, ACS & American Society for the Control of Cancer.
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TL;DR: It is suggested that prediagnosis BMI, but not post diagnosis BMI, is an important predictor of survival among patients with nonmetastatic colorectal cancer.
Abstract: Purpose The impact of body mass index (BMI) on survival after colorectal cancer diagnosis is poorly understood. This study assessed the association of pre- and postdiagnosis BMI with all-cause and cause-specific survival among men and women diagnosed with colorectal cancer in a prospective cohort. Patients and Methods Participants in the Cancer Prevention Study-II Nutrition Cohort reported weight and other risk factor information via a self-administered questionnaire at baseline in 1992 to 1993. Updated information on current weight and incident cancer was reported via periodic follow-up questionnaires. This analysis includes 2,303 cohort participants who were diagnosed with nonmetastatic colorectal cancer between baseline and mid 2007 and were observed for mortality from diagnosis through December 2008. Results A total of 851 participants with colorectal cancer died during the 16-year follow-up period, including 380 as a result of colorectal cancer and 153 as a result of cardiovascular disease (CVD). In ...
176 citations
Johns Hopkins University1, Harvard University2, Yale University3, National Institutes of Health4, German Cancer Research Center5, New York University6, Mayo Clinic7, University of Toronto8, University of California, San Francisco9, International Agency for Research on Cancer10, University of Pisa11, Sapienza University of Rome12, Vita-Salute San Raffaele University13, Leidos14, University of Amsterdam15, Yale Cancer Center16, Veterans Health Administration17, National and Kapodistrian University of Athens18, Monash University19, Cancer Council Victoria20, University of Melbourne21, Fred Hutchinson Cancer Research Center22, Heidelberg University23, University of Southern California24, University of Texas MD Anderson Cancer Center25, University of Szeged26, Charles University in Prague27, American Cancer Society28, University of Ostrava29, University of Cambridge30, Cleveland Clinic31, Lithuanian University of Health Sciences32, Memorial Sloan Kettering Cancer Center33, University of Hawaii34, Carlos III Health Institute35, National Institute for Health and Welfare36, QIMR Berghofer Medical Research Institute37, University of Padua38, University of Bologna39, Autonomous University of Barcelona40, Pompeu Fabra University41, Université Paris-Saclay42, Vanderbilt University43, Georgetown University44, Umeå University45, Casa Sollievo della Sofferenza46, Kaiser Permanente47, University of Hamburg48, Academy of Sciences of the Czech Republic49, University at Buffalo50, University of Washington51
TL;DR: The largest pancreatic cancer GWAS to date, including 9040 patients and 12,496 controls of European ancestry from the PANScan and the Pancreatic Cancer Case Control Consortium (PanC4), was performed in this paper.
Abstract: In 2020, 146,063 deaths due to pancreatic cancer are estimated to occur in Europe and the United States combined. To identify common susceptibility alleles, we performed the largest pancreatic cancer GWAS to date, including 9040 patients and 12,496 controls of European ancestry from the Pancreatic Cancer Cohort Consortium (PanScan) and the Pancreatic Cancer Case-Control Consortium (PanC4). Here, we find significant evidence of a novel association at rs78417682 (7p12/TNS3, P = 4.35 × 10−8). Replication of 10 promising signals in up to 2737 patients and 4752 controls from the PANcreatic Disease ReseArch (PANDoRA) consortium yields new genome-wide significant loci: rs13303010 at 1p36.33 (NOC2L, P = 8.36 × 10−14), rs2941471 at 8q21.11 (HNF4G, P = 6.60 × 10−10), rs4795218 at 17q12 (HNF1B, P = 1.32 × 10−8), and rs1517037 at 18q21.32 (GRP, P = 3.28 × 10−8). rs78417682 is not statistically significantly associated with pancreatic cancer in PANDoRA. Expression quantitative trait locus analysis in three independent pancreatic data sets provides molecular support of NOC2L as a pancreatic cancer susceptibility gene.
176 citations
TL;DR: Current ACS cancer screening guidelines are summarized, as are the latest data on the use of cancer screening from the National Health Interview Survey, in this issue of the journal.
Abstract: Each year the American Cancer Society (ACS) publishes a summary of its recommendations for early cancer detection, a report on data and trends in cancer screening rates, and select issues related to cancer screening. In this issue of the journal, current ACS cancer screening guidelines are summarized, as are the latest data on the use of cancer screening from the National Health Interview Survey.
176 citations
TL;DR: The data suggest that very low intakes of plant foods may increase risk, and that certain phytochemical subgroups may decrease risk in men and women through August 31, 1997.
Abstract: Objective: We examined the relation between whole grains, fruit, vegetables and dietary fiber and colon cancer risk in the prospective Cancer Prevention Study II Nutrition Cohort. Methods: In 1992–1993, 62,609 men and 70,554 women completed questionnaires on medical history, diet and lifestyle behaviors. After exclusions, we confirmed 298 cases of incident colon cancer among men and 210 among women through August 31, 1997. Results: Multivariate rate ratios (RR) and 95% confidence intervals (CI) for all dietary factors were null. However, a statistically non-significant 30% reduction in risk was observed for men with the highest vegetable intakes (RR = 0.69, CI = 0.47–1.03, top versus. bottom quintile, p trend = 0.10). Men with very low (lowest tertile within the lowest quintile) intakes of vegetables and dietary fiber were at increased risk compared to those in the highest four quintiles of intake (vegetables RR = 1.79, CI = 1.22–2.61, p trend = 0.04, and fiber RR = 1.96, CI = 1.24–3.10, p trend = 0.006). Women with very low intakes of fruit were also at increased risk (RR = 1.86, CI =1.18–2.94, p trend = 0.06). Conclusions: Higher intakes of plant foods or fiber were not related to lower risk of colon cancer. However, our data suggest that very low intakes of plant foods may increase risk, and that certain phytochemical subgroups may decrease risk.
176 citations
TL;DR: In this paper, the authors use the literature to develop conceptual and working definitions of ECB and use these to present a frame for reading the four case studies, and use them to develop a conceptual framework for reading case studies.
Abstract: The authors use the literature to develop conceptual and working definitions of ECB and use these to present a frame for reading the four case studies.
175 citations
Authors
Showing all 1345 results
| Name | H-index | Papers | Citations |
|---|---|---|---|
| Walter C. Willett | 334 | 2399 | 413322 |
| Meir J. Stampfer | 277 | 1414 | 283776 |
| Frank B. Hu | 250 | 1675 | 253464 |
| David J. Hunter | 213 | 1836 | 207050 |
| Edward Giovannucci | 206 | 1671 | 179875 |
| Irving L. Weissman | 201 | 1141 | 172504 |
| Bernard Rosner | 190 | 1162 | 147661 |
| Susan E. Hankinson | 151 | 789 | 88297 |
| Paolo Boffetta | 148 | 1455 | 93876 |
| Jeffrey A. Bluestone | 143 | 515 | 77080 |
| Richard D. Smith | 140 | 1180 | 79758 |
| Garth D. Illingworth | 137 | 505 | 61793 |
| Brian E. Henderson | 137 | 712 | 69921 |
| Ahmedin Jemal | 132 | 500 | 380474 |
| Michael J. Thun | 129 | 392 | 79051 |