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Beata Swiatkowska

Researcher at Nofer Institute of Occupational Medicine

Publications -  32
Citations -  1581

Beata Swiatkowska is an academic researcher from Nofer Institute of Occupational Medicine. The author has contributed to research in topics: Medicine & Cancer. The author has an hindex of 11, co-authored 17 publications receiving 1108 citations.

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Journal ArticleDOI

Obesity, metabolic factors and risk of different histological types of lung cancer: A Mendelian randomization study

Robert Carreras-Torres, +64 more
- 08 Jun 2017 - 
TL;DR: The results are consistent with a causal role of fasting insulin and low-density lipoprotein cholesterol in lung cancer etiology, as well as for BMI in squamous cell and small cell carcinoma, and the latter relation may be mediated by a previously unrecognized effect of obesity on smoking behavior.
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Large-scale association analysis identifies new lung cancer susceptibility loci and heterogeneity in genetic susceptibility across histological subtypes

James D. McKay, +146 more
- 12 Jun 2017 - 
TL;DR: 18 susceptibility loci achieving genome-wide significance are identified, including 10 new loci linked with lung cancer overall and six loci associated with lung adenocarcinoma, highlighting the striking heterogeneity in genetic susceptibility across the histological subtypes of lung cancer.
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Genome-wide association study identifies eight risk loci and implicates metabo-psychiatric origins for anorexia nervosa

Hunna J. Watson, +209 more
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Identification of susceptibility pathways for the role of chromosome 15q25.1 in modifying lung cancer risk

Xuemei Ji, +133 more
TL;DR: Functional annotation of eQTL analysis results showed that the neuroactive ligand receptor interaction pathway and gated channel activity were involved in lung cancer risk, and provide important insights for the etiology of lung cancer.
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Fine mapping of MHC region in lung cancer highlights independent susceptibility loci by ethnicity.

Aida Ferreiro-Iglesias, +74 more
TL;DR: Genetic variation within the MHC region in lung cancer patients and independent associations within HLA genes that explain MHC lung cancer associations in Europeans and Asian populations are considered.