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Simon C. Watkins

Researcher at University of Pittsburgh

Publications -  999
Citations -  75771

Simon C. Watkins is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Apoptosis & Immune system. The author has an hindex of 135, co-authored 950 publications receiving 68358 citations. Previous affiliations of Simon C. Watkins include Harvard University & Children's National Medical Center.

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Journal ArticleDOI

A quantitative comparison of satellite cell ultrastructure in Duchenne muscular dystrophy, polymyositis, and normal controls.

TL;DR: The ultrastructural composition of the satellite cells in 25 DMD cases is examined and they are compared with satellite cells from normal subjects and from polymyositis patients to try to identify morphological features that might be associated with an impaired developmental competence.
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Nitric oxide-induced modification of protein thiolate clusters as determined by spectral fluorescence resonance energy transfer in live endothelial cells

TL;DR: In this paper, spectral laser scanning confocal imaging was used to visualize the effects of D- and L-stereoisomers of S-nitrosocysteine ethyl ester (SNCEE) on fluorescence resonance energy transfer (FRET)-based reporters that are targets for the following NO-related modifications: (a) S -nitrosation, via the cysteine-rich protein metallothionein, and (b) nitrosyl-heme-Fe, via guanosine 3',5'-cyclic monophosph
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An investigation of fluttering piezoelectric energy harvesters in off-axis and turbulent flows

TL;DR: In this paper, the response of a fluttering piezoelectric energy harvester was studied in smooth flow and in aspects of replicated Atmospheric Boundary Layer turbulence (12.7% intensity, 310mm longitudinal integral length scale).
Proceedings ArticleDOI

Application of Smart Materials for Adaptive Airfoil Shape Control

TL;DR: This investigation is the first step in the development of an adaptive airfoil that is able to be utilized on an Uninhibited Aerial Vehicle and the influence of changingAirfoil’s maximum camber on aerodynamic performance was explored and the analysis is presented.
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Hepatocyte Fas-associating death domain protein/mediator of receptor-induced toxicity (FADD/MORT1) levels increase in response to pro-apoptotic stimuli.

TL;DR: The interesting and unexpected result that actinomycin D increased the expression of FADD protein is reported, and it is demonstrated that other cellular stresses like ultraviolet irradiation or heat shock could also increase FADD levels in hepatocytes.