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Robert C. Thompson

Researcher at University of Michigan

Publications -  142
Citations -  26223

Robert C. Thompson is an academic researcher from University of Michigan. The author has contributed to research in topics: Receptor & Gene expression. The author has an hindex of 62, co-authored 140 publications receiving 23428 citations. Previous affiliations of Robert C. Thompson include Nagoya University & Molecular and Behavioral Neuroscience Institute.

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Global, regional, and national disability-adjusted life-years (DALYs) for 333 diseases and injuries and healthy life expectancy (HALE) for 195 countries and territories, 1990–2016: a systematic analysis for the Global Burden of Disease Study 2016

Simon I. Hay, +803 more
- 16 Sep 2017 - 
TL;DR: At a global level, DALYs and HALE continue to show improvements and the importance of continued health interventions, which has changed in most locations in pace with the gross domestic product per person, education, and family planning.
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Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs

S. Hong Lee, +405 more
- 01 Sep 2013 - 
TL;DR: Empirical evidence of shared genetic etiology for psychiatric disorders can inform nosology and encourages the investigation of common pathophysiologies for related disorders.
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Analysis of shared heritability in common disorders of the brain

Verneri Anttila, +720 more
- 22 Jun 2018 - 
TL;DR: It is demonstrated that, in the general population, the personality trait neuroticism is significantly correlated with almost every psychiatric disorder and migraine, and it is shown that both psychiatric and neurological disorders have robust correlations with cognitive and personality measures.
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Large-scale genome-wide association analysis of bipolar disorder identifies a new susceptibility locus near ODZ4

Pamela Sklar, +192 more
- 01 Oct 2011 - 
TL;DR: An analysis of all 11,974 bipolar disorder cases and 51,792 controls confirmed genome-wide significant evidence of association for CACNA1C and identified a new intronic variant in ODZ4, and a pathway comprised of subunits of calcium channels enriched in bipolar disorder association intervals was identified.
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Interleukin-1 receptor antagonist activity of a human interleukin-1 inhibitor

TL;DR: Three interleukin-1 inhibitors have been purified to homogeneity from medium conditioned by human monocytes and partial sequence analysis and digestion with N-glycanase indicate that these are glycosylation forms of a single protein.