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Michael Karin

Researcher at University of California, San Diego

Publications -  753
Citations -  246120

Michael Karin is an academic researcher from University of California, San Diego. The author has contributed to research in topics: IκB kinase & Signal transduction. The author has an hindex of 236, co-authored 704 publications receiving 226485 citations. Previous affiliations of Michael Karin include Sanford-Burnham Institute for Medical Research & University of California, Los Angeles.

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Casein kinase II is a negative regulator of c-Jun DNA binding and AP-1 activity.

TL;DR: One of the roles of CKII, a major nuclear protein kinase with no known functions, is to attenuate AP-1 activity through phosphorylation of c-Jun, a phosphoprotein in nonstimulated fibroblasts and epithelial cells.
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c-Fos transcriptional activity stimulated by H-Ras-activated protein kinase distinct from JNK and ERK

TL;DR: RAS proteins exert their mitogenic and oncogenic effects through activation of downstream protein kinases, and at least three types of proline-directed kinases transmit Ras- and mitogen-generated signals to the transcriptional machinery.
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Stabilization of interleukin-2 mRNA by the c-Jun NH2-terminal kinase pathway.

TL;DR: Signaling pathways that stabilize interleukin-2 (IL-2) messenger RNA (mRNA) in activated T cells were examined and multiple elements within IL-2 mRNA modulate its stability in a combinatorial manner.
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Control of transcription factors by signal transduction pathways: the beginning of the end.

TL;DR: The mechanisms that control the activity of two groups of sequence-specific transcription factors, the AP-1 and CREB/ATF proteins, are described and serve as a paradigm explaining the transfer of regulatory information from the cell surface to the nucleus.
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Three distinct signalling responses by murine fibroblasts to genotoxic stress

TL;DR: Findings show that signals generated by genotoxins are transduced by multiple, independent pathways, and only p53 appears to be a universal sensor of genotoxic stress.