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Helen Ferry

Researcher at University of Oxford

Publications -  34
Citations -  1917

Helen Ferry is an academic researcher from University of Oxford. The author has contributed to research in topics: B cell & Antigen. The author has an hindex of 18, co-authored 31 publications receiving 1558 citations. Previous affiliations of Helen Ferry include John Radcliffe Hospital & University College London.

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Journal ArticleDOI

Guidelines for the use of flow cytometry and cell sorting in immunological studies (second edition)

Andrea Cossarizza, +462 more
- 01 Oct 2019 - 
TL;DR: These guidelines are a consensus work of a considerable number of members of the immunology and flow cytometry community providing the theory and key practical aspects offlow cytometry enabling immunologists to avoid the common errors that often undermine immunological data.
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Persistent malignant stem cells in del(5q) myelodysplasia in remission.

Ramin Tehranchi, +20 more
- 08 Sep 2010 - 
TL;DR: Rare and phenotypically distinct del(5q) myelodysplastic syndrome stem cells were identified that were also selectively resistant to therapeutic targeting at the time of complete clinical and cytogenetic remission.
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The earliest thymic T cell progenitors sustain B cell and myeloid lineage potential.

Sidinh Luc, +37 more
- 01 Apr 2012 - 
TL;DR: This work provides functional and molecular evidence at the single-cell level that the earliest progenitors in the neonatal thymus had combined granulocyte-monocyte, T lymphocyte and B lymphocyte lineage potential but not megakaryocyte-erythroid lineage potential.
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Cited2 Is an Essential Regulator of Adult Hematopoietic Stem Cells

Kamil R. Kranc, +10 more
- 04 Dec 2009 - 
TL;DR: Using loss-of-function approaches, genetic evidence is provided that Cited2 selectively maintains adult HSC functions, at least in part, via Ink4a/Arf and Trp53.
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CD4 T cell-dependent autoimmunity against a melanocyte neoantigen induces spontaneous vitiligo and depends upon Fas-Fas ligand interactions.

Teresa Lambe, +8 more
- 01 Sep 2006 - 
TL;DR: The findings show that CD4-dependent destruction of melanocytes is partially inhibited by blocking Fas-Fas ligand interactions and also highlights the importance of local control of autoimmunity, as vitiligo remains patchy and never proceeds to confluence even when Ag and autoreactive CD4+ T cells are abundant.