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Jens Geginat

Researcher at University of Milan

Publications -  81
Citations -  13623

Jens Geginat is an academic researcher from University of Milan. The author has contributed to research in topics: Cytotoxic T cell & Cytokine. The author has an hindex of 38, co-authored 72 publications receiving 11984 citations. Previous affiliations of Jens Geginat include Charité.

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Journal ArticleDOI

Central Memory and Effector Memory T Cell Subsets: Function, Generation, and Maintenance

Federica Sallusto, +2 more
- 19 Mar 2004 - 
TL;DR: This review addresses the heterogeneity of TCM and TEM, their differentiation stages, and the current models for their generation and maintenance in humans and mice.
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Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells

Eva V. Acosta-Rodríguez, +7 more
- 01 Jun 2007 - 
TL;DR: It is demonstrated that human TH-17 cells have distinct migratory capacity and antigenic specificities and a link between microbial products, T helper cell differentiation and homing in response to fungal antigens is established.
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Biology of interleukin-10

Robert Sabat, +6 more
- 01 Oct 2010 - 
TL;DR: The special physiological relevance of this cytokine lies in the prevention and limitation of over-whelming specific and unspecific immune reactions and, in consequence, of tissue damage.
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Guidelines for the use of flow cytometry and cell sorting in immunological studies (second edition)

Andrea Cossarizza, +462 more
- 01 Oct 2019 - 
TL;DR: These guidelines are a consensus work of a considerable number of members of the immunology and flow cytometry community providing the theory and key practical aspects offlow cytometry enabling immunologists to avoid the common errors that often undermine immunological data.
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Th17 cells transdifferentiate into regulatory T cells during resolution of inflammation

Nicola Gagliani, +22 more
- 09 Jul 2015 - 
TL;DR: Two new fate-mapping mouse models are used to show that CD4+ T cells that formerly expressed IL-17A go on to acquire an anti-inflammatory phenotype and suggest that Th17 cell instability and plasticity is a therapeutic opportunity for inflammatory diseases.