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Carlo M. Croce

Researcher at Ohio State University

Publications -  1156
Citations -  199822

Carlo M. Croce is an academic researcher from Ohio State University. The author has contributed to research in topics: microRNA & Cancer. The author has an hindex of 198, co-authored 1135 publications receiving 189007 citations. Previous affiliations of Carlo M. Croce include University of Nebraska Medical Center & University of California, Los Angeles.

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Journal Article

Genetic alterations of the tumor suppressor gene WWOX in esophageal squamous cell carcinoma.

TL;DR: Results indicate that alteration and inactivation of the WWOX gene may play a role in esophageal squamous cell carcinogenesis.
Journal ArticleDOI

microRNA Involvement in Hepatocellular Carcinoma

TL;DR: Functional and molecular studies uncovered mechanisms that linked deregulated miRNAs to cancer-associated pathways, thereby placing their deregulation in a more rational framework and improving knowledge concerning the molecular basis of HCC.
Journal Article

Amplification of the c-myc Oncogene in One of Five Human Breast Carcinoma Cell Lines

Danuta Kozbor, +1 more
- 01 Feb 1984 - 
TL;DR: The c-myc oncogene was not rearranged in any of five different human breast carcinoma cell lines examined, and a 10-fold amplification and an elevated expression of the oncogen were detected in one of these cell lines.
Journal ArticleDOI

BCL2-mediated tumorigenicity of a human T-lymphoid cell line: synergy with MYC and inhibition by BCL2 antisense.

TL;DR: Observations extend to T cells the finding of synergy of BCL2 with MYC previously reported for B cells and provide evidence that BCL1 can regulate the growth of human T cells.
Journal Article

Characterization of the 13q14 tumor suppressor locus in CLL: identification of ALT1, an alternative splice variant of the LEU2 gene.

TL;DR: A segment of 790 kb at 13q14 spanning the minimal region of loss in CLL is completely sequenced and characterized, finding that this feature of the sequence may favor the occurrence of chromosomal rearrangements and may confer instability to the region, resulting in deletions that may inactivate an as yet unidentified tumor suppressor.