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Carlo M. Croce

Researcher at Ohio State University

Publications -  1156
Citations -  199822

Carlo M. Croce is an academic researcher from Ohio State University. The author has contributed to research in topics: microRNA & Cancer. The author has an hindex of 198, co-authored 1135 publications receiving 189007 citations. Previous affiliations of Carlo M. Croce include University of Nebraska Medical Center & University of California, Los Angeles.

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Prostate specific antigen in breast cancer, benign breast disease and normal breast tissue

TL;DR: Examining quantitatively the PSA levels in 199 breast tumors, 48 tissues with benign breast disease, and 36 normal breast tissues suggests that PSA is expressed frequently by normal breast tissue, by tissue of benign breast diseases, and by breast cancer tissue.
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MicroRNA expression profiling in human Barrett's carcinogenesis

TL;DR: In this article, a miRNA microarray analysis comparing native esophageal mucosa with all the phenotypic lesions seen in the Barrett's carcinogenic process was performed to test the hypothesis that a specific miRNA expression signature characterizes BM development and progression.
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Loss of Heterozygosity at Chromosome 11q in Lung Adenocarcinoma: Identification of Three Independent Regions

TL;DR: The findings suggest the presence of at least three tumor suppressor genes on the long arm of chromosome 11, and confirm the relevance of 11q22-24, a region frequently deleted in carcinomas of the breast, ovary, uterine, cervix, colon, and malignant melanoma in the pathogenesis of solid tumors.
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Chemoprevention of Cigarette Smoke–Induced Alterations of MicroRNA Expression in Rat Lungs

TL;DR: MicroRNA analysis may provide a new tool for predicting at early carcinogenesis stages both the potential safety and efficacy of cancer chemopreventive agents.
Journal Article

Loss of FHIT Expression in Gastric Carcinoma

TL;DR: The consequent absence or reduction of Fhit protein expression is consistent with the proposal that the FHIT gene is a preferential target of environmental carcinogens and that FHit inactivation plays a role in the development of gastric cancer.