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David Voehringer

Researcher at University of Erlangen-Nuremberg

Publications -  127
Citations -  10280

David Voehringer is an academic researcher from University of Erlangen-Nuremberg. The author has contributed to research in topics: Immune system & Immunoglobulin E. The author has an hindex of 41, co-authored 111 publications receiving 8481 citations. Previous affiliations of David Voehringer include University of California, San Francisco & University of Freiburg.

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Journal ArticleDOI

The transcription factor GATA-3 controls cell fate and maintenance of type 2 innate lymphoid cells.

Thomas Hoyler, +8 more
- 19 Oct 2012 - 
TL;DR: It is demonstrated that GATA-3 is essential for ILC2 fate decisions and similarities between the transcriptional programs controlling ILC and T helper cell fates are revealed.
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Chitin induces accumulation in tissue of innate immune cells associated with allergy

Tiffany A. Reese, +7 more
- 03 May 2007 - 
TL;DR: It is shown that chitin induces the accumulation in tissue of IL-4-expressing innate immune cells, including eosinophils and basophils, when given to mice, and this process can be negatively regulated by a vertebrate chit inase.
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Guidelines for the use of flow cytometry and cell sorting in immunological studies (second edition)

Andrea Cossarizza, +462 more
- 01 Oct 2019 - 
TL;DR: These guidelines are a consensus work of a considerable number of members of the immunology and flow cytometry community providing the theory and key practical aspects offlow cytometry enabling immunologists to avoid the common errors that often undermine immunological data.
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The microbiota regulates type 2 immunity through RORγt+ T cells

Caspar Ohnmacht, +22 more
- 28 Aug 2015 - 
TL;DR: It is reported that microbiota-induced Tregs express the nuclear hormone receptor RORγt and differentiate along a pathway that also leads to TH17 cells, and acts as a key factor in balancing immune responses at mucosal surfaces.
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Constitutive ablation of dendritic cells breaks self-tolerance of CD4 T cells and results in spontaneous fatal autoimmunity

Caspar Ohnmacht, +6 more
- 16 Mar 2009 - 
TL;DR: It is shown that DCs play an essential role to protect against fatal autoimmunity under steady-state conditions and could be induced by reconstitution of wild-type mice with bone marrow from ΔDC mice, whereas mixed BM chimeras that received BM from Δ DC and WT mice remained healthy.