Institution
University of Alabama
Education•Tuscaloosa, Alabama, United States•
About: University of Alabama is a education organization based out in Tuscaloosa, Alabama, United States. It is known for research contribution in the topics: Population & Poison control. The organization has 27323 authors who have published 48609 publications receiving 1565337 citations. The organization is also known as: Alabama & Bama.
Topics: Population, Poison control, Galaxy, Health care, Large Hadron Collider
Papers published on a yearly basis
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TL;DR: 1-Ethyl-3-methyl-imidazolium acetate can completely dissolve raw crustacean shells, leading to recovery of a high purity, high molecular weight chitin powder and to fibers and films which can be spun directly from the extract solution.
353 citations
University of Alabama1, International Agency for Research on Cancer2, University of California, San Francisco3, Ohio State University4, Medical Research Council5, University of Manchester6, University of Cambridge7, University of Antwerp8, University of Ljubljana9, Queen Mary University of London10, National Institutes of Health11, Centers for Disease Control and Prevention12, University of Western Ontario13, University of New South Wales14, University of Cape Town15, McGill University16, VU University Amsterdam17, Harvard University18
TL;DR: There must be ongoing efforts including international advocacy to achieve widespread-optimally universal-implementation of HPV prevention strategies in both developed and developing countries.
352 citations
TL;DR: In this article, the authors trace the evolution of right-wing extremism, conceptualized as latent electoral support for extreme rightwing parties (i.e., vote intention), in six Western European countries between 1984 and 1993.
Abstract: This study traces the evolution of right-wing extremism, conceptualized as latent electoral support for extreme right-wing parties (i.e., vote intention), in six Western European countries (i.e., Belgium, France, the Netherlands, West Germany, Denmark, and Italy) between 1984 and 1993. Employing a pooled time-series cross-sectional research design, the author examines the relative strength of three popular explanations of contemporary right-wing extremism: the impact of economic conditions (unemployment and inflation), social developments (immigration), and political trends (public's dissatisfaction with the political regime). Evidence is presented in support of the last two explanations. Rising levels of immigration and public dissatisfaction with the political regime significantly facilitate right-wing extremism. Contrary to the initial hypothesis however, results suggest that a declining national economy (unemployment in particular) diminishes the electoral appeal of extreme right-wing parties.
351 citations
TL;DR: An N-ethyl-N-nitrosourea–induced mutant mouse, alien (aln), is described, which has abnormal primary cilia and shows overactivation of the SHH pathway, suggesting that anterograde IFT is required for GLI activation and that retrograde IFT modulates this event.
Abstract: Characterization of previously described intraflagellar transport (IFT) mouse mutants has led to the proposition that normal primary cilia are required for mammalian cells to respond to the sonic hedgehog (SHH) signal. Here we describe an N-ethyl-N-nitrosourea–induced mutant mouse, alien (aln), which has abnormal primary cilia and shows overactivation of the SHH pathway. The aln locus encodes a novel protein, THM1 (tetratricopeptide repeat–containing hedgehog modulator-1), which localizes to cilia. aln-mutant cilia have bulb-like structures at their tips in which IFT proteins (such as IFT88) are sequestered, characteristic of Chlamydomonas reinhardtii and Caenorhabditis elegans retrograde IFT mutants. RNA-interference knockdown of Ttc21b (which we call Thm1 and which encodes THM1) in mouse inner medullary collecting duct cells expressing an IFT88–enhanced yellow fluorescent protein fusion recapitulated the aln-mutant cilial phenotype, and live imaging of these cells revealed impaired retrograde IFT. In contrast to previously described IFT mutants, Smoothened and full-length glioblastoma (GLI) proteins localize to aln-mutant cilia. We hypothesize that the aln retrograde IFT defect causes sequestration of IFT proteins in aln-mutant cilia and leads to the overactivated SHH signaling phenotype. Specifically, the aln mutation uncouples the roles of anterograde and retrograde transport in SHH signaling, suggesting that anterograde IFT is required for GLI activation and that retrograde IFT modulates this event.
351 citations
TL;DR: In this article, the authors examined three research questions using recent sample data: (1) Is Altman's original Z-score model as useful for predicting bankruptcy in recent periods as it was for the periods in which it was developed and tested by Altman? (2) Is the model as good for predicting non-manufacturing firms as it is for predicting manufacturing firms? (3) Is it as good as the model for predicting financial stress conditions other than bankruptcy, as well as predicting bankruptcy?
349 citations
Authors
Showing all 27508 results
| Name | H-index | Papers | Citations |
|---|---|---|---|
| Jasvinder A. Singh | 176 | 2382 | 223370 |
| Hongfang Liu | 166 | 2356 | 156290 |
| Ian J. Deary | 166 | 1795 | 114161 |
| Yongsun Kim | 156 | 2588 | 145619 |
| Dong-Chul Son | 138 | 1370 | 98686 |
| Simon C. Watkins | 135 | 950 | 68358 |
| Kenichi Hatakeyama | 134 | 1731 | 102438 |
| Conor Henderson | 133 | 1387 | 88725 |
| Peter R Hobson | 133 | 1590 | 94257 |
| Tulika Bose | 132 | 1285 | 88895 |
| Helen F Heath | 132 | 1185 | 89466 |
| James Rohlf | 131 | 1215 | 89436 |
| Panos A Razis | 130 | 1287 | 90704 |
| David B. Allison | 129 | 836 | 69697 |
| Eduardo Marbán | 129 | 579 | 49586 |