Institution
Tokyo University of Science
Education•Tokyo, Japan•
About: Tokyo University of Science is a education organization based out in Tokyo, Japan. It is known for research contribution in the topics: Catalysis & Thin film. The organization has 15800 authors who have published 24147 publications receiving 438081 citations. The organization is also known as: Tōkyō Rika Daigaku & Science University of Tokyo.
Papers published on a yearly basis
Papers
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TL;DR: A facile aqueous acid treatment on the flame-made BiVO(4) is introduced, which in the presence of small addition of Bi and V promotes full transformation to scheelite-monoclinic and reduces charge trapping defects, and the photocatalytic O(2) evolution activity was increased by a remarkable 5 folds.
Abstract: Visible-light-active BiVO4 photocatalyst prepared by a one-step flame spray pyrolysis demonstrates the structural evolution from amorphous to crystalline scheelite-tetragonal and further to scheelite-monoclinic (the photocatalytic active phase). Up to 95% scheelite-monoclinic content, the rest being scheelite-tetragonal, can be achieved in situ by exposing the collection filter to higher flame temperature. Reasonable activity in terms of photocatalytic O2 evolution was obtained with the increase in crystallinity and scheelite-monoclinic content. Although analogous postcalcination of BiVO4 improves crystallization and phase transformation, it inevitably induces vacancy defects that are detrimental to the photocatalytic activity. Hence a facile aqueous acid treatment on the flame-made BiVO4 is introduced, which in the presence of small addition of Bi and V promotes full transformation to scheelite-monoclinic and reduces charge trapping defects. As a result, the photocatalytic O2 evolution activity was incre...
127 citations
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TL;DR: Fluorescence spectra and powder X-ray diffraction patterns demonstrated that pyrene molecules are captured in an amorphous state in the range of low pyrene content (<5%), while excimer formation was seen at the higher pyrene concentration (>5%).
127 citations
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TL;DR: In this article, the formation mechanism and the origin of the two-dimensional electron gas (2DEG) in ZnMgO/ZnO heterostructures have been investigated.
Abstract: Both the formation mechanism and the origin of the two-dimensional electron gas (2DEG) in ZnMgO/ZnO heterostructures have been investigated. The 2DEG in the heterostructures was confirmed to originate from polarization-induced charge and was found to be dominant for transport at low temperatures as well as room temperature (RT) by transport measurements. The origin of 2DEG was concluded to be the surface of the ZnMgO layer based on both capacitance-voltage measurements and the dependence of the carrier concentration on the ZnMgO layer thickness. The largest sheet carrier concentration was 1.1×1013 cm−2 and the highest mobility for the heterostructure was obtained for a Mg composition of 0.61 at RT.
126 citations
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TL;DR: In this article, a combination system consisting of a H2 production photocatalyst, Pt/SrTiO3:Rh, and an O2 O2 production copacalyst, BiVO4 or WO3, decomposed water into H2 and O2 under visible light irradiation in...
Abstract: A combination system consisting of a H2 production photocatalyst, Pt/SrTiO3:Rh, and an O2 production photocatalyst, BiVO4 or WO3, decomposed water into H2 and O2 under visible light irradiation in ...
126 citations
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TL;DR: The findings suggest that IL-1 receptor antagonist deficiency in T cells disrupts homeostasis of the immune system and that TNF-alpha plays an important role in activating T cells through induction of OX40.
Abstract: IL-1 receptor antagonist-deficient (IL-1Ra(-/-)) mice spontaneously develop autoimmune arthritis. We demonstrate here that T cells are required for the induction of arthritis; T cell-deficient IL-1Ra(-/-) mice did not develop arthritis, and transfer of IL-1Ra(-/-) T cells induced arthritis in nu/nu mice. Development of arthritis was also markedly suppressed by TNF-alpha deficiency. We found that TNF-alpha induced OX40 expression on T cells and blocking the interaction between either CD40 and its ligand or OX40 and its ligand suppressed development of arthritis. These findings suggest that IL-1 receptor antagonist deficiency in T cells disrupts homeostasis of the immune system and that TNF-alpha plays an important role in activating T cells through induction of OX40.
126 citations
Authors
Showing all 15878 results
Name | H-index | Papers | Citations |
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Kazunori Kataoka | 138 | 908 | 70412 |
Yoichiro Iwakura | 129 | 705 | 64041 |
Kouji Matsushima | 124 | 590 | 56995 |
Masaki Ishitsuka | 103 | 624 | 39383 |
Shinsuke Tanabe | 98 | 722 | 37445 |
Tatsumi Koi | 97 | 411 | 50222 |
Hirofumi Akagi | 94 | 618 | 43179 |
Clifford A. Lowell | 91 | 258 | 23538 |
Teruo Okano | 91 | 605 | 28346 |
László Á. Gergely | 89 | 426 | 60674 |
T. Sumiyoshi | 88 | 855 | 62277 |
Toshinori Nakayama | 86 | 405 | 25275 |
Akihiko Kudo | 86 | 328 | 39475 |
Hans-Joachim Gabius | 85 | 699 | 28085 |
Motohide Tamura | 85 | 1007 | 32725 |