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The 2016 revision of the World Health Organization classification of lymphoid neoplasms

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TLDR
The revision clarifies the diagnosis and management of lesions at the very early stages of lymphomagenesis, refines the diagnostic criteria for some entities, details the expanding genetic/molecular landscape of numerous lymphoid neoplasms and their clinical correlates, and refers to investigations leading to more targeted therapeutic strategies.
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This article is published in Blood.The article was published on 2016-05-19 and is currently open access. It has received 5321 citations till now.

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Epidemiology, genetics and treatment of multiple myeloma and precursor diseases.

TL;DR: In this paper, the authors summarized the current understanding of multiple myeloma including its epidemiology, genetics and biology, and provided an overview of MM management that have led to improvements in survival.
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Usefulness of CD200 in the differential diagnosis of SDRPL, SMZL, and HCL

TL;DR: The expression of this marker could improve the specificity and/or sensitivity of the current scoring system in SDRPL diagnosis and very recent detection of recurrent mutations in those entities may now help to further assess the differential diagnosis of SDR PL, SMZL, and HCL.
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Molecular background delineates outcome of double protein expressor diffuse large B-cell lymphoma

TL;DR: The findings define molecular characteristics of the DPE in DLBCL, and recognize clinically feasible predictors of outcome, and identify non-GCB lymphomas with favorable BN2/C1-like survival independent of IPI and concurrent DPE status.
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Mogamulizumab: An Anti-CC Chemokine Receptor 4 Antibody for T-Cell Lymphomas.

TL;DR: The recent approval of mogamulizumab represents an important addition to the armamentarium of pharmacotherapies for T-cell lymphomas, providing a new option in the treatment of relapsed or refractory cutaneous T- cell lymphomas.
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Classical Hodgkin's Lymphoma in the Era of Immune Checkpoint Inhibition

TL;DR: This review provides in this review the recent findings focused on the major research topics, such as adverse events and resistance to PD-1–PD-L1 inhibitor treatment, together with a part about angiogenesis, extracellular vesicles, and microbiome in HL pathogenesis.
References
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Book

WHO Classification of Tumours of Haematopoietic and Lymphoid Tissues

TL;DR: Thank you very much for reading who classification of tumours of haematopoietic and lymphoid tissues, and maybe you have knowledge that, people have look hundreds of times for their chosen readings like this, but end up in malicious downloads.
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The 2008 WHO classification of lymphoid neoplasms and beyond: evolving concepts and practical applications.

TL;DR: The criteria and significance of early or precursor lesions and the identification of certain lymphoid neoplasms largely associated with particular age groups, such as children and the elderly are addressed, and the issue of borderline categories having overlapping features with large B-cell lymphomas is reviewed.
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Whole-genome sequencing identifies recurrent mutations in chronic lymphocytic leukaemia

TL;DR: The patterns of somatic mutation, supported by functional and clinical analyses, strongly indicate that the recurrent NOTCH1, MYD88 and XPO1 mutations are oncogenic changes that contribute to the clinical evolution of the disease.
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MYD88 L265P Somatic Mutation in Waldenström's Macroglobulinemia

TL;DR: MYD88 L265P is a commonly recurring mutation in patients with Waldenström's macroglobulinemia that can be useful in differentiating WaldenStröm’s macrogalobulinesia and non-IgM LPL from B-cell disorders that have some of the same features.
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