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The 2016 revision of the World Health Organization classification of lymphoid neoplasms

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TLDR
The revision clarifies the diagnosis and management of lesions at the very early stages of lymphomagenesis, refines the diagnostic criteria for some entities, details the expanding genetic/molecular landscape of numerous lymphoid neoplasms and their clinical correlates, and refers to investigations leading to more targeted therapeutic strategies.
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This article is published in Blood.The article was published on 2016-05-19 and is currently open access. It has received 5321 citations till now.

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Characterization of the fecal microbiome in cats with inflammatory bowel disease or alimentary small cell lymphoma.

TL;DR: Feline chronic enteropathy in cats showed patterns of dysbiosis similar to those in found people with IBD, and no significant difference between the microbiome of cats with I BD and those with SCL was found.
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CD79B mutations in primary vitreoretinal lymphoma: Diagnostic and prognostic potential.

TL;DR: The mutation in PVRL was examined to clarify its diagnostic and prognostic potential and to clarify the development of central nervous system (CNS) lesions during its course.
References
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WHO Classification of Tumours of Haematopoietic and Lymphoid Tissues

TL;DR: Thank you very much for reading who classification of tumours of haematopoietic and lymphoid tissues, and maybe you have knowledge that, people have look hundreds of times for their chosen readings like this, but end up in malicious downloads.
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The 2008 WHO classification of lymphoid neoplasms and beyond: evolving concepts and practical applications.

TL;DR: The criteria and significance of early or precursor lesions and the identification of certain lymphoid neoplasms largely associated with particular age groups, such as children and the elderly are addressed, and the issue of borderline categories having overlapping features with large B-cell lymphomas is reviewed.
Journal ArticleDOI

Whole-genome sequencing identifies recurrent mutations in chronic lymphocytic leukaemia

TL;DR: The patterns of somatic mutation, supported by functional and clinical analyses, strongly indicate that the recurrent NOTCH1, MYD88 and XPO1 mutations are oncogenic changes that contribute to the clinical evolution of the disease.
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MYD88 L265P Somatic Mutation in Waldenström's Macroglobulinemia

TL;DR: MYD88 L265P is a commonly recurring mutation in patients with Waldenström's macroglobulinemia that can be useful in differentiating WaldenStröm’s macrogalobulinesia and non-IgM LPL from B-cell disorders that have some of the same features.
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