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Rakesh K. Jain

Researcher at Harvard University

Publications -  1528
Citations -  198912

Rakesh K. Jain is an academic researcher from Harvard University. The author has contributed to research in topics: Angiogenesis & Cancer. The author has an hindex of 200, co-authored 1467 publications receiving 177727 citations. Previous affiliations of Rakesh K. Jain include Government Medical College, Thiruvananthapuram & University of Oslo.

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Book ChapterDOI

Development of Deep UV LEDs and Current Problems in Material and Device Technology

TL;DR: In this article, the authors review progress in development of deep ultraviolet light-emitting diodes and discuss key factors currently affecting device performance, and discuss the importance of alloy composition fluctuations based on results of time-resolved temperature-dependent photoluminescence and lightinduced grating measurements of Al0.6Ga0.4N layers with different density of dislocations.
Journal ArticleDOI

Selectin ligands: 2,3,4-tri-O-acetyl-6-O-pivaloyl-α/β-galactopyranosyl halide as novel glycosyl donor for the synthesis of 3-O-sialyl or 3-O-sulfo Lexand Leatype structures

TL;DR: Stereoselective syntheses of 3-O-sialyl- and 3-Sulfo- Lewisx and Lewisa type structures are accomplished by the use of key glycosyl donors 8 and 9.
Journal ArticleDOI

Effects of cediranib (VEGF signaling inhibitor) on edema in newly diagnosed glioblastoma patients during initial chemoradiation.

TL;DR: In patients with newly diagnosed GBM treated with CRT and cediranib, tumor volume decreased on T1 and FLAIR images whereas the FLAIR volume significantly increased in the control group suggesting increased edema.
Journal ArticleDOI

The asparagine residue in the FRNK box of potyviral helper-component protease is critical for its small RNA binding and subcellular localization.

TL;DR: In vitro studies suggest that amino acid N(182) of the conserved FRNK box may regulate RNA silencing mechanisms, and is required for maintenance of the subcellular localization of the protein for its multi-functionality.
Journal ArticleDOI

Tospo viral infection instigates necrosis and premature senescence by micro RNA controlled programmed cell death in Vigna unguiculata

TL;DR: To understand the mechanistic model of symptom development and the role of programmed cell death (PCD) during infection, the levels of biochemical intermediates and stress responsive micro RNAs (miRNAs) with their target transcripts in Vigna unguiculata are compared.