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Christian Hofmann

Researcher at University of California, Los Angeles

Publications -  10
Citations -  1048

Christian Hofmann is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: CTL* & Cytotoxic T cell. The author has an hindex of 5, co-authored 10 publications receiving 747 citations. Previous affiliations of Christian Hofmann include University of Erlangen-Nuremberg.

Papers
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Journal ArticleDOI

Rapid Decay of Anti–SARS-CoV-2 Antibodies in Persons with Mild Covid-19

F. Javier Ibarrondo, +9 more
- 21 Jul 2020 - 
TL;DR: Covid-19 Antibodies after Mild Infection Among 34 volunteers who had recovered from mild Covid- 19 illness, antiviral antibodies to the receptor-binding domain of the viral spike protein declined.
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Primary, Recall, and Decay Kinetics of SARS-CoV-2 Vaccine Antibody Responses.

F. Javier Ibarrondo, +16 more
- 23 Jun 2021 - 
TL;DR: In this paper, a longitudinal assessment of vaccinees with and without prior SARS-CoV-2 infection using quantitative enzyme-linked immunosorbent assay (ELISA) of anti-RBD antibodies was conducted.
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Proceedings of the Frontiers of Retrovirology Conference 2016

Irena Zurnic, +659 more
- 26 Sep 2016 - 
TL;DR: The present work has presented a novel single-cell analysis pipeline to identify specific biomarkers of HIV permissiveness and the importance of the tax-inducible actin-bundling protein fascin for transmission of human T cell leukemia virus Type 1 (HTLV-1).
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Humoral responses to SARS-CoV-2 mRNA vaccines: Role of past infection.

Ashley Gray, +17 more
- 08 Nov 2021 - 
TL;DR: In this article, two mRNA vaccines (BNT162b2 and mRNA-1273) against severe acute respiratory syndrome-coronavirus 2 (SARS-CoV-2) are globally authorized as a two-dose regimen.
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HIV-1 epitopes presented by MHC class I types associated with superior immune containment of viremia have highly constrained fitness landscapes.

Aleksandr M. Gorin, +9 more
- 07 Aug 2017 - 
TL;DR: Overall, these findings indicate that these protective MHC-I types yield CTL targeting of highly constrained epitopes, and underscore the importance of blocking public escape pathways for CTL-based interventions against HIV-1.