Journal ArticleDOI
Role and mechanisms of cytokines in the secondary brain injury after intracerebral hemorrhage.
Huimin Zhu,Zhiqiang Wang,Jixu Yu,Xiuli Yang,Feng He,Zhenchuan Liu,Fengyuan Che,Xuemei Chen,Honglei Ren,Michael Hong,Jian Wang +10 more
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TLDR
A review summarizes the roles and functions of various pro- and anti-inflammatory cytokines in secondary brain injury after ICH and discusses pathogenic mechanisms and emerging therapeutic strategies and directions for treatment of ICH.About:
This article is published in Progress in Neurobiology.The article was published on 2019-07-01. It has received 162 citations till now. The article focuses on the topics: Proinflammatory cytokine & Intracerebral hemorrhage.read more
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Caspase-1-induced pyroptosis is an innate immune effector mechanism against intracellular bacteria (111.33)
Edward A. Miao,Irina A. Leaf,Piper M. Treuting,Dat P. Mao,Anasuya Sarkar,Mark D. Wewers,Alan Aderem +6 more
TL;DR: It is demonstrated that activation of caspase-1 clears intracellular bacteria in vivo independently of IL-1β and IL-18 and establishes pyroptosis as an efficient mechanism of bacterial clearance by the innate immune system.
Journal ArticleDOI
Neuroinflammation in intracerebral haemorrhage: immunotherapies with potential for translation.
Mengzhou Xue,V. Wee Yong +1 more
TL;DR: The quick initiation of these drugs, particularly in high systemic doses, could be key to counteracting the evolving secondary injury in people with intracerebral haemorrhage and provides a promising way in which the poor prognosis of intrACErebralHaemorhage might one day be counteracted.
Journal ArticleDOI
Potential therapeutic targets for intracerebral hemorrhage-associated inflammation: An update
TL;DR: An update of recent progress on ICH is provided and the prospects for further preclinical studies in this field are described and new therapeutic targets and directions for the treatment of ICH are discussed.
Journal ArticleDOI
Pathophysiological Mechanisms and Potential Therapeutic Targets in Intracerebral Hemorrhage.
Zhiwei Shao,Sheng Tu,Anwen Shao +2 more
TL;DR: Some key pathophysiology mechanisms in ICH such as oxidative stress, inflammation, iron toxicity, and thrombin formation are discussed and the corresponding therapeutic targets and therapeutic strategies are reviewed.
Journal ArticleDOI
Recombinant Adiponectin Peptide Ameliorates Brain Injury Following Intracerebral Hemorrhage by Suppressing Astrocyte-Derived Inflammation via the Inhibition of Drp1-Mediated Mitochondrial Fission.
Xun Wu,Jianing Luo,Haixiao Liu,Wenxing Cui,Kang Guo,Lei Zhao,Hao Bai,Wei Guo,Hao Guo,Dayun Feng,Yan Qu +10 more
TL;DR: First mechanistic evidence is provided that APNp can modulate Drp1-mediated mitochondrial fission, which then contributes to alleviating astrocyte-derived inflammation, ultimately attenuating inflammatory brain injury induced by hemorrhage.
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Piel,Dan Poenaru,Suzanne Polinder,Svetlana Popova,Richie Poulton,Farshad Pourmalek,Dorairaj Prabhakaran,Dima M. Qato,Amado D Quezada,D. Alex Quistberg,Felicia A. Rabito,Anwar Rafay,Kazem Rahimi,Vafa Rahimi-Movaghar,Sajjad Ur Rahman,Murugesan Raju,Ivo Rakovac,Saleem M Rana,Saleem M Rana,Amany H Refaat,Giuseppe Remuzzi,Antonio Luiz Pinho Ribeiro,Stefano Ricci,Patricia M. Riccio,Lee Richardson,Jan Hendrik Richardus,Bayard Roberts,D. Allen Roberts,Margaret Robinson,Anna Roca,Alina Rodriguez,David Rojas-Rueda,Luca Ronfani,Robin Room,Gregory A. Roth,Dietrich Rothenbacher,David H. Rothstein,Jane Rowley,Nobhojit Roy,George Mugambage Ruhago,Lesley Rushton,Sankar Sambandam,Kjetil Søreide,Mohammad Yahya Saeedi,Sukanta Saha,Ramesh Sahathevan,Mohammad Ali Sahraian,Berhe W. Sahle,Joshua A. Salomon,Deborah Salvo,Genesis May J. Samonte,Uchechukwu K.A. Sampson,Juan Sanabria,Logan Sandar,Itamar S. 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Warouw,Scott Weichenthal,Elisabete Weiderpass,Robert G. Weintraub,Robert G. Weintraub,Wang Wenzhi,Andrea Werdecker,K. Ryan Wessells,Ronny Westerman,Harvey Whiteford,James D. Wilkinson,Thomas N. Williams,Solomon Meseret Woldeyohannes,Charles D.A. Wolfe,Timothy M. Wolock,Anthony D. Woolf,John Q. Wong,Jonathan L. Wright,Sarah Wulf,Brittany Wurtz,Gelin Xu,Yang Yang,Yuichiro Yano,Hiroshi Yatsuya,Paul S. F. Yip,Naohiro Yonemoto,Seok Jun Yoon,Mustafa Z. Younis,Chuanhua Yu,Kim Yun Jin,Maysaa El Sayed Zaki,Mohammed Fouad Zamakhshary,Hajo Zeeb,Yong Zhang,Yong Zhao,Yingfeng Zheng,Jun Zhu,Shankuan Zhu,David Zonies,Xiao Nong Zou,Joseph R. Zunt,Theo Vos,Alan D. Lopez,Alan D. Lopez,Christopher J L Murray,Gabriel Alcalá-Cerra,Howard Hu,Nadim E. Karam,Nsanzimana Sabin,A. M. Temesgen +731 more
TL;DR: In the Global Burden of Disease Study 2013 (GBD 2013) as discussed by the authors, the authors used the GBD 2010 methods with some refinements to improve accuracy applied to an updated database of vital registration, survey, and census data.
Journal ArticleDOI
IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
Claire L. Langrish,Yi Yi Chen,Wendy M. Blumenschein,Jeanine D. Mattson,Beth Basham,Jonathan D. Sedgwick,Terrill K. McClanahan,Robert A. Kastelein,Daniel J. Cua +8 more
TL;DR: Using passive transfer studies, it is confirmed that these IL-23–dependent CD4+ T cells are highly pathogenic and essential for the establishment of organ-specific inflammation associated with central nervous system autoimmunity.
Journal ArticleDOI
Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes.
TL;DR: The results indicate that IL-10 has important regulatory effects on immunological and inflammatory responses because of its capacity to downregulate class II MHC expression and to inhibit the production of proinflammatory cytokines by monocytes.
Journal ArticleDOI
Bcl-2 antiapoptotic proteins inhibit Beclin 1-dependent autophagy.
Sophie Pattingre,Sophie Pattingre,Amina T. Tassa,Xueping Qu,Xueping Qu,Rita Garuti,Xiao Huan Liang,Noboru Mizushima,Milton Packer,Michael D. Schneider,Beth Levine,Beth Levine +11 more
TL;DR: Bcl-2 not only functions as an antiapoptotic protein, but also as an antiautophagy protein via its inhibitory interaction with Beclin 1, which may help maintain autophagy at levels that are compatible with cell survival, rather than cell death.
Journal ArticleDOI
IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines
Jochen Schmitz,Alexander Owyang,Elizabeth R. Oldham,Yaoli Song,Erin Murphy,Terril K. McClanahan,Gerard Zurawski,Mehrdad M. Moshrefi,Jinzhong Qin,Xiaoxia Li,Daniel M. Gorman,J. Fernando Bazan,Robert A. Kastelein +12 more
TL;DR: A member of theIL-1 family, IL-33, which mediates its biological effects via IL-1 receptor ST 2, activates NF-kappaB and MAP kinases, and drives production of T(H)2-associated cytokines from in vitro polarized T( H)2 cells is reported.