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Ryan P. Hillary

Researcher at Stanford University

Publications -  11
Citations -  1001

Ryan P. Hillary is an academic researcher from Stanford University. The author has contributed to research in topics: Genome-wide association study & Cancer. The author has an hindex of 6, co-authored 10 publications receiving 621 citations.

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The NASA Twins Study: A multidimensional analysis of a year-long human spaceflight.

Francine E. Garrett-Bakelman, +88 more
- 12 Apr 2019 - 
TL;DR: Given that the majority of the biological and human health variables remained stable, or returned to baseline, after a 340-day space mission, these data suggest that human health can be mostly sustained over this duration of spaceflight.
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Association Between Telomere Length and Risk of Cancer and Non-Neoplastic Diseases A Mendelian Randomization Study

Philip C Haycock, +197 more
- 01 May 2017 - 
TL;DR: It is likely that longer telomeres increase risk for several cancers but reduce risk for some non-neoplastic diseases, including cardiovascular diseases, as well as single nucleotide polymorphisms (SNPs) that are strongly associated with telomere length in the general population.
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Kleine-Levin syndrome is associated with birth difficulties and genetic variants in the TRANK1 gene loci

TL;DR: The authors found a strong genome-wide significant association (rs71947865, Odds Ratio [OR] = 1.48, P = 8.6 × 10-9) within the 3'gion of TRANK1 gene locus, previously associated with bipolar disorder and schizophrenia.
Posted ContentDOI

Narcolepsy risk loci are enriched in immune cells and suggest autoimmune modulation of the T cell receptor repertoire

Hanna Ollila, +87 more
- 22 Jul 2018 - 
TL;DR: Together these findings show that the autoimmune component in narcolepsy is defined by antigen presentation, mediated through specific T cell receptor chains, and modulated by influenza-A as a critical trigger.
Journal ArticleDOI

Complex HLA association in paraneoplastic cerebellar ataxia with anti-Yo antibodies.

TL;DR: High resolution HLA typing of Class I/Class II in 40 cases versus ethnically matched controls suggests differential genetic susceptibility to anti-Yo per cancer and with a primary HLA Class II involvement.