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Alistair R. R. Forrest

Researcher at Harry Perkins Institute of Medical Research

Publications -  184
Citations -  27204

Alistair R. R. Forrest is an academic researcher from Harry Perkins Institute of Medical Research. The author has contributed to research in topics: Gene & Regulation of gene expression. The author has an hindex of 59, co-authored 175 publications receiving 23544 citations. Previous affiliations of Alistair R. R. Forrest include Griffith University & Centre for Life.

Papers
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The Transcriptional Landscape of the Mammalian Genome

Piero Carninci, +197 more
- 02 Sep 2005 - 
TL;DR: Detailed polling of transcription start and termination sites and analysis of previously unidentified full-length complementary DNAs derived from the mouse genome provide a comprehensive platform for the comparative analysis of mammalian transcriptional regulation in differentiation and development.
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An atlas of active enhancers across human cell types and tissues

TL;DR: It is shown that enhancers share properties with CpG-poor messenger RNA promoters but produce bidirectional, exosome-sensitive, relatively short unspliced RNAs, the generation of which is strongly related to enhancer activity.
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A promoter-level mammalian expression atlas

Alistair R. R. Forrest, +280 more
- 27 Mar 2014 - 
TL;DR: For example, the authors mapped transcription start sites (TSSs) and their usage in human and mouse primary cells, cell lines and tissues to produce a comprehensive overview of mammalian gene expression across the human body.
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Analysis of the mouse transcriptome based on functional annotation of 60,770 full-length cDNAs

Yasushi Okazaki, +138 more
- 05 Dec 2002 - 
TL;DR: The present work, completely supported by physical clones, provides the most comprehensive survey of a mammalian transcriptome so far, and is a valuable resource for functional genomics.
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Genome-wide analysis of mammalian promoter architecture and evolution

TL;DR: These tagging methods allow quantitative analysis of promoter usage in different tissues and show that differentially regulated alternative TSSs are a common feature in protein-coding genes and commonly generate alternative N termini.