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Journal ArticleDOI

A colorimetric method for the determination of serum glutamic oxalacetic and glutamic pyruvic transaminases.

Stanley Reitman, +1 more
- 01 Jul 1957 - 
- Vol. 28, Iss: 1, pp 56-63
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This article is published in American Journal of Clinical Pathology.The article was published on 1957-07-01. It has received 9424 citations till now.

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Journal ArticleDOI

Liver damage in heatstroke.

TL;DR: The effects of heatstroke on the structure and function of the liver have been studied in Bantu gold miners and it is suggested that the hepatic changes are the result of a combination of hypoxia and direct thermal injury.
Journal ArticleDOI

Protective effect of ursolic acid on ethanol-mediated experimental liver damage in rats

TL;DR: Ursolic acid improved the antioxidant status of alcoholic rats, which is evaluated by the decreased levels of lipid peroxidation markers in plasma and increased levels of circulatory antioxidants such as reduced glutathione, ascorbic acid and alpha-tocopherol.
Journal ArticleDOI

Effect of Garlic Oil on the Levels of Various Enzymes in the Serum and Tissue of Streptozotocin Diabetic Rats

TL;DR: Levels of red cell, serum acid, and alkaline phosphatases, serum amylase, alanine and aspartate transferase and bilirubin were examined in streptozotocin-induced diabetic rats treated with garlic oil and compared with the corresponding levels in diabetic control rats, normal rats and normal rats on garlic oil.
Journal ArticleDOI

Serum Creatine Phosphokinase (CPK) Activity in Disorders of Heart and Skeletal Muscle.

TL;DR: Serum enzyme determinations that are commonly available in hospital laboratories for aid in the diagnosis of diseases of the heart and skeletal muscle lack tissue specificity, so it may be that these determinations are inappropriate for use in clinical practice.
Journal ArticleDOI

Glutathione metabolizing enzymes and oxidative stress in ferric nitrilotriacetate mediated hepatic injury.

TL;DR: It is indicated that treatment with Fe-NTA eventuates in decreased hepatic GSH, a fall in the activities of glutathione metabolizing enzymes and excessive production of oxidants, all of which are involved in the cascade of events leading to iron-mediated hepatic injury.
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