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Ajit Varki

Researcher at University of California, San Diego

Publications -  557
Citations -  63836

Ajit Varki is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Sialic acid & SIGLEC. The author has an hindex of 124, co-authored 542 publications receiving 58772 citations. Previous affiliations of Ajit Varki include Emory University & National Institute of Advanced Industrial Science and Technology.

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Genetic Differences Between Humans and Great Apes -- Implications for the Evolution of Humans

Ajit Varki
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Abstract 411: Human Species-specific Loss of the CMP-N-acetylneuraminic Acid Hydroxylase Fuels Atherosclerosis Development via Intrinsic and Extrinsic Mechanisms

Kunio Kawanishi, +6 more
- 01 May 2018 - 
TL;DR: The objective of this study was to establish a baseline level of baseline atherosclerosis in mice that is consistent with that in other mammals and show clear patterns of progression towards atherosclerotic disease.
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Steps in the formation of asparagine-linked carbohydrate moieties

Stuart Kornfeld, +3 more
- 01 Apr 1981 - 
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Program and abstracts for the 2012 Joint Meeting of the Society for Glycobiology & American Society for Matrix Biology

Ronald L. Schnaar, +1604 more
- 01 Nov 2012 - 
TL;DR: Time 8:30 Glycobiology of Human Pluripotent Stem Cells; Steve Dalton, University of Georgia #1; Siglec-Sialoglycan Binding Regulates Cell-Cell Interactions; Ronald Schnaar, Johns Hopkins School of Medicine.
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Multiple Hepatic Clearance Receptors Facilitate Elimination of Secretory Mucins that Can Enter the Bloodstream in Pathological States and Generate Platelet-Rich Microthrombi.

Mark G. Wahrenbrock, +1 more
- 16 Nov 2004 - 
TL;DR: Multiple hepatic clearance receptors collaborate to varying degrees to ensure elimination of the many polydisperse and differentially glycosylated forms of secretory mucins that might enter the blood circulation from various hollow organs, thus limiting their ability to cause pathology involving disseminated platelet and leukocyte aggregation.