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Pascale Cossart

Researcher at Pasteur Institute

Publications -  438
Citations -  54204

Pascale Cossart is an academic researcher from Pasteur Institute. The author has contributed to research in topics: Listeria monocytogenes & Internalin. The author has an hindex of 124, co-authored 434 publications receiving 50101 citations. Previous affiliations of Pascale Cossart include University of British Columbia & University of Paris.

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Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Comparative Genomics of Listeria Species

Philippe Glaser, +54 more
- 26 Oct 2001 - 
TL;DR: A large number of predicted genes encoding surface and secreted proteins, transporters, and transcriptional regulators are found, consistent with the ability of both species to adapt to diverse environments.
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Bacterial invasion: the paradigms of enteroinvasive pathogens.

Pascale Cossart, +1 more
- 09 Apr 2004 - 
TL;DR: The mechanisms underlying bacterial entry, phagosome maturation, and dissemination reveal common strategies as well as unique tactics evolved by individual species to establish infection.
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E-Cadherin Is the Receptor for Internalin, a Surface Protein Required for Entry of L. monocytogenes into Epithelial Cells

J Mengaud, +4 more
- 22 Mar 1996 - 
TL;DR: It is reported the first identification of a cellular receptor mediating entry of a gram-positive bacterium into nonphagocytotic cells by an affinity chromatography approach, and reveals a novel type of heterophilic interactions for E-cadherin.
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Bacterial Adhesion and Entry into Host Cells

Javier Pizarro-Cerdá, +5 more
- 24 Feb 2006 - 
TL;DR: Bacteria use monomeric adhesins/invasins or highly sophisticated macromolecular machines to establish a complex host/pathogen molecular crosstalk that leads to subversion of cellular functions and establishment of disease.